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Interesting

Hormones support angiogenesis. Angiogenesis is stimulated when tumor tissues require nutrients and oxygen. It's quite a complicated process when in the context of oncology.
 
Presumably something that suppresses angiogensis would also improve outcomes? So slowing or preventing a tumor improving it's blood supply and then growing. I'm guessing this is also like the Ivor Cummings thread, ie specific types of tumors. I'm also guessing in this case, it's helped by the blood-brain barrier. So if the brain and tumor prefers glucose, forcing ketosis means the brain keeps going, but the tumor may be starved.
 
Thalidomide has been used in oncology trials because of its anti-angiogenesis property. Other drugs are used - of course it all depends on the cell type of the tumour.
You have a good 'handle' on this, if I can be so bold.

In my opinion, the overriding crux of the tumour/glucose debate, must rest with the knowledge that all rapidly dividing cells have an increased glucose utilisation, but, the amount is likely to be a minuscule amount in comparison to a 4g teaspoon of glucose.
 
Lots of cansor grow slower if inslin levels are lower, I expect that metformin is working more by reducing inslin levels then BG levels.

I think in a trail on metform less people died from censor then expected, so it is now being investigated. The tail was not looking at cansor, hence the data was not clear.

If our body can kill cansor cells then just slowing down the cansor growth rate may allow our body to kill it in some cases.
 
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