bulkbiker
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- Messages
- 19,575
- Type of diabetes
- Type 2
- Treatment type
- Diet only
Well surely it is in excess when caused by an insulinoma..insulin release is not a disease
You can hardly claim that is the "normal" way of things?
Well surely it is in excess when caused by an insulinoma..insulin release is not a disease
You could have high insulin levels but not have a hypo and in such a case we wouldn't see this drive to eat - why not if it is insulin?
Regarding your arthritic knee analogy. The cause of the pain is the disease - insulin release is not a disease and only can be considered in this context if there is a pathological condition either driving abnormal release of it or an abnormal response to it. The arthritic knee hurts when you walk but insulin doesn't automatically cause hunger unless it causes a hypo which happens within the context of certain/another condition. I fail to see how you would not see this distinction as important even if treatment may be the same even if you don't.
Actually, again drawing on personal experience is very useful in this, because I can tell you that unless I walk, or move, in a manner that results in pain, I am pain free - therefore your statement that the cause of the pain is the disease is inaccurate.
The pain arises from moving in a way which aggravates existing damage, or generates more damage, not from the arthritis itself.
Anyway, this is a circular argument which, while very entertaining, is unlikely to result in either of us changing our opinions, and while pedantry runs strongly in my family, it is a rabbit hole I try not to indulge in too often.
I very much look forward to your references to support the claims you have made in previous posts.
Well surely it is in excess when caused by an insulinoma..
You can hardly claim that is the "normal" way of things?
The study indicates that reducing blood sugars in a way that mimicked the so called post meal blood sugar spike and crash did not stimulate appetite. 1 study but an interesting one seeming to confirm that in the absence of a frank hypo spikes/drops in blood sugar do not stimulate a desire to eat. https://www.sciencedirect.com/science/article/abs/pii/S0195666316302604
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They are both simply incorrect.
Possibly because people are more concerned with putting a "chronic, progressive " disease into remission as opposed to athletic performance?
In your not so humble opinion I guess.. his name is Dr Paul Mason by the way.
So what field of health care are you in exactly?
Thank you for the reference, unfortunately, it has no relevance to this discussion.
The title of the study clearly states that the participants were healthy men. Therefore, comparisons cannot be drawn between either the individual who had an insulinoma, or with people who have excess insulin production, and/or insulin resistance - which you yourself (in your next post) describe as 'a pathological condition'.
One thing I most definitely agree with you about, is that you and I should stop discussing my arthritic knee.
Your comments on the subject are very far from the mark, and show some major misunderstandings about arthritis, its progression, and how to 'fix' it.
In your not so humble opinion I guess.. his name is Dr Paul Mason by the way.
So what field of health care are you in exactly?
All, this thread is about Ivor Cummins and Keto. If you wish to discuss sporting performance and low carb/keto or other aspects related to diet and appetite, please do so on another thread. Thanks.
Independent of calories insulin will not make you fat
It is relevant...
I'm hearing insulin and glucose discussed. What about leptin? How do high and low levels of insulin affect leptin, and appetite?