The insulin resistance you mention may well be adaptive glucose sparing (physiological resistance rather than pathological). To be noted it goes away within days of reintroducing carbs. I would guess for a type 1 insulin dosing purposes the difference between different types of IR is immaterial.The other thing I have read (but did not continue the diet to experience it myself) is that a low/no carb diet increases insulin resistance so you may be more basal.
Interesting. As it's only been a few weeks I need to give it time. Definitely much better control already, I just assumed I'd need hardly any short acting fiasp as I'm eating very few carbs.My dietician told me that I would need higher insulin ratios for a low (less than 100g) diet. I believe (possibly incorrectly) that my total insulin dose is less than I would need for a higher carb diet.
Still not counting protein but possibly I should?
I have a differet insulin ratio for mornings when I eat breakfast and mornings when I don't...
Health line.com discusses their keto diet findings (although they do not cite their sources for this) on mice. I quote “They found that keto diets don’t allow the body to properly use insulin, so blood sugar isn’t properly controlled. That leads to insulin resistance” ……… Which I’m guessing is what you are saying @HSSS .The insulin resistance you mention may well be adaptive glucose sparing (physiological resistance rather than pathological). To be noted it goes away within days of reintroducing carbs. I would guess for a type 1 insulin dosing purposes the difference between different types of IR is immaterial.
Where it does matter is in type 2 where pathological IR is our main issue. This specific temporary form of IR doesn’t cause damage in the way the more typical one does and is done to preserve the essential small amounts of glucose, the brain and a few other processes absolutely require, in the absence of all but minimal carbs. It can mean slightly raised fasted readings but maintains good responses to keto meals and a flatter line all day resulting in fewer peaks and troughs and better hba1 ultimately
edited for clarity
I’m not saying glucose isn’t properly controlled. I’m saying glucose gets used differently than the accepted “norm on a carb heavy diet”. Insulin resistance comes in different ”flavours”, not all are problematic. I’m not a mouse. Perhaps it depends on the definition of “properly”Health line.com discusses their keto diet findings (although they do not cite their sources for this) on mice. I quote “They found that keto diets don’t allow the body to properly use insulin, so blood sugar isn’t properly controlled. That leads to insulin resistance” ……… Which I’m guessing is what you are saying @HSSS .
@Mflannagan have you been diagnosed T1 late onset? If you have then LADA is known to have some similarities with may T2. Insulin resistance maybe present.
Ha, no you are not a mouse @HSSS , not as far as I’m aware!I’m not saying glucose isn’t properly controlled. I’m saying glucose gets used differently than the accepted “norm on a carb heavy diet”. Insulin resistance comes in different ”flavours”, not all are problematic. I’m not a mouse. Perhaps it depends on the definition of “properly”
This one ? https://pubmed.ncbi.nlm.nih.gov/18054972/@HSSS thanks for the mention about IR coming in ‘different flavours’. I found this piece on IR
‘Insulin resistance in an energy-centered perspective’, Ping Wang, Edwin C M Mariman, 2008.
I couldn't attach the link as I’m not at home.
I had no idea there are positives associated with IR .
ed - spelling
That’s the one @HSSS .
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