LADA

tarredweet said:

The reason for this is that I was not overweight at the time of diagnosis and kept myself fit.

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You do not have to be overweight, nor unfit, to develop T2 diabetes. I cannot comment on your specific case -- it may be a misdiagnosis, that is always possible. But weight is unlikely to be the clinching piece of evidence.

Hi. Being slim is one indicator of T1 together with blood sugar that can't be controlled with all the usual tablets. Sudden unexpected weight loss adds to the possibility of LADA. I've been down the LADA route myself. I had the two tests for it done privately. C-peptide which measures your insulin is a good indicator. GAD tests for antibodies. A negative GAD does not prove you aren't LADA; mine was negative and in my case I suspect a virus. There are those who say LADA has to be due to antibody attack. I don't agree with that view and would say that any destruction of the beta cells makes you T1 (LADA) as the cause doesn't matter - the result and treatment is the same. So, make sure your HBA1C is acceptable within NICE guidelines and if it's too high ask the GP for the two tests. Are you on various tablets?

I’m LADA. 100% agree with @Daibell Wright loss is a big indicator. I too think my ‘ progression’ was from a virus. Doesn’t matter. I have a very low c peptide and high GAD. Those are tests are the on,y way to determine or not. LADA requires insulin as we don’t make any. T2 using insulin COULD POSSIBLY insulin resistance controlled by diet and exercise.

tarredweet said:

No,I am on insulin and have been since two months after my diagnosis.I started off on Metformin then progressed to insulin

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Hi. It sounds like you are in the same state as me i.e. listed as T2 but 'wrongly' and should be LADA (or based on some recent posts T3c even! There is a big problem with the NHS and DUK having a very rigid (and very silly) view that beta cell damage only results from antibodies. This is classed as T1 and anything else is labelled T2. T2 is therefore used as a dumping ground for a complete range of conditions where the treatments may be very different due to under or over production of insulin. I argued with my GP that I was not T2 but T1 and she arrogantly told me she was the expert and refused me insulin as I was T2. A year later she offered me insulin when my HBa1C went thru the roof and asked me why my HBa1C had suddenly shot up. I resisted the temptation to tell her it was 'cos I was T1 and my insulin production was dying. I believe in 5 to 10 years time the classing of types will be radically changed and diagnosis based more around insulin production rather than causes. This should lead to better treatment and lead to better statistics for research as the current NHS stats don't reflect the 10-15% 'T2s' who are slim and in fact LADA.