NickiSanta
Member
- Messages
- 17
- Type of diabetes
- Prediabetes
- Treatment type
- Diet only
This study utilised intensive medical (drug) treatments and possibly the lower levels associated with hypos rather than stable normal levels and potentially side effects of medication as well as likely underlying increasing insulin resistance. As such surely that’s quite different to non medical methods (like low carb and increased insulin sensitivity that results) so not especially relevant to a non medicated approach.Unless you have weight to lose (adipose fat / abdominal spare tyre) then going into ketosis is probably unecessary. There have been studies done that show that running with sugars below 5 mmol.l is not actually beneficial and the mortality curve starts to rise below 5.4 mmol/l. My doctor prefers me to have a target of 7 mmol/l and my hospital consultant advises 12 mmol/l as his recommendation. Needless to say I set my eyes on about 6 and am currently running an average of 6.4.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2258341/
A valid observation. But looking at the overall T2D cohort, the position at the moment is that the majority are following current NICE guidelines and Eatwell or worse. The number of people using a sensible diet to control their condition is definitely a minority. There are also reports coming out that Low Carb diets are also associated with increased mortality, but not increased CVD outcomes. LCHF in its current form is a recent introduction, and does not have long term studies to support its safety yet. We here are guinea pigs in that respect. We have yet to really see the implications of that diet in the long term. There is longer experience and review of restricted calorie low fat diets for instance, and some of them have been shown to be injurious. Most of the diet research is connected to the Health and Fitness industry, which forms the basis of our Nutrition theory. The weight loss industry is also involved but do not fund research to any significant level. The medical use of diets and research do not really seem to team up except in Eatwell.This study utilised intensive medical (drug) treatments and possibly the lower levels associated with hypos rather than stable normal levels and potentially side effects of medication as well as likely underlying increasing insulin resistance. As such surely that’s quite different to non medical methods (like low carb and increased insulin sensitivity that results) so not especially relevant to a non medicated approach.
In fact the study itself says “This leaves us with the intervention itself as the possible culprit—the highly intensive treatment (often multiple insulin injection regimens combined with multiple oral agents) aimed at reaching the target of HbA1c <6.0%.” and “The problem of increased deaths may in some way be linked to higher doses of injected insulin in combination with stimulation of endogenous insulin secretion (by sulfonylureas) or use of insulin sensitisers (metformin and thiazolidinediones), or both. If insensitivity to insulin is actually a protective mechanism, rather than the pathological outcome of overeating as it is perceived today, then perhaps trying aggressively to overcome it may have adverse cellular effects that we have not yet begun to understand.”
But we are talking - here in this thread - about low carb not the general cohort so again I make the point this study is not particularly relevant to this specific conversation.A valid observation. But looking at the overall T2D cohort, the position at the moment is that the majority are following current NICE guidelines and Eatwell or worse. The number of people using a sensible diet to control their condition is definitely a minority. There are also reports coming out that Low Carb diets are also associated with increased mortality, but not increased CVD outcomes. LCHF in its current form is a recent introduction, and does not have long term studies to support its safety yet. We here are guinea pigs in that respect. We have yet to really see the implications of that diet in the long term. There is longer experience and review of restricted calorie low fat diets for instance, and some of them have been shown to be injurious. Most of the diet research is connected to the Health and Fitness industry, which forms the basis of our Nutrition theory. The weight loss industry is also involved but do not fund research to any significant level. The medical use of diets and research do not really seem to team up except in Eatwell.
Whilst it is difficult to see why a low carb approach should be associated with an increase in mortlity since all it is doing is mimicing 'normal' non diabetic range of bgl, it is nonetheless an intervention which may be inducing its own co-morbidities. We do not at the moment know for certain that keeping bgl in target range as a normal non diabetic actually reduces all the complications linked to T2 diabetes. I am hopefull that it does, but we see evidence from bariatric surgery follow up that it is not the complete picture. The remission is not a cure, and it is not pemanent.
You are putting the blinkers on. The paper is not apparently relevant to the OP but may well apply to readers of the thread. Your point is valid, but there is still open ground around it that other papers show similar stories and outcome. The curve is a bathtub curve, and while different medications affect the actual inflection point, there will come a stage where the curve changes slope and rise and going too low seems to be harmful. even in ketosis, our brains and nerves need glucose, which we synthesise from protein if necessary. but that is an inefficient process, and is limited to provide the essential functions for survival. ketosis is actually a form of starvation, and yes, we do appear to be able to sustain a lifestyle with it, but only while there is fat in lipid form to use for overall energy. As I said above, the keto diet is a new kid on the block, and we need to see what happens as it reaches maturity. as an ex-smoker, I am indeed paying the price now in later life.But we are talking - here in this thread - about low carb not the general cohort so again I make the point this study is not particularly relevant to this specific conversation.
Bariatric surgery is a different approach to low carb and it’s successes and failures are once again not the same thing as other methods of control and it’s results should not be extrapolated directly across to low carb methods, even whilst there may be links between them.
Please link the reports that low carb alone is increasing mortality or inducing co-morbidities. This claim is quite alarming to see and needs supporting
I accept that low carb to treat diabetes is a new approach on any sort of widespread scale. Nor are we convinced yet that it does reduce all the complications but it certainly is at least helping many of them so far. Maybe we won’t stop every sign and symptom in the long term but it seems to be doing an awful lot better than purely medication approaches.
No one here, and certainly not I, is suggesting cure.
I'd say you don't have bg problems but you are adjusting from having higher bgs to lower ones and to burning of fat for fuel rather than glucose from food. You can't have a true hypo (insulin over dose) unless you are taking blood sugar lowering meds or insulin itself (that doesn't include metformin and most other types of diabetic meds).Thank you for your replies
My carb intake is very low (less than 22g a day), and yes I'm probably in ketosis. I didn't know I could lower my blood sugar enough to feel ill just by diet alone. :O
I guess that means water fasting is out of the question. I'd heard it can reset all sorts of things including blood glucose problems.
One aspect to consider is that current home testers have an error allowance or _/- 0.7 mmol/l at the levels you are measuring and would still be considered as 'accurate'. i.e. meeting their specification. Hypo's technically start when the bgl drops below 3.9 mmol/l. so your 3.2 could be the start of a minor hypo, but is possibly a 3.9 masquerading as a hypo. either way, a couple of jelly babies or a slice of bread will sort that out.Hi
I got a pre-diabetes diagnosis in December and after reading and researching I decided to alter my eating to low carb.
I test before and after meals and my fasting level.
I've managed to get my readings into the 4s and 5s, but several times a week I catch my BG in the 3s . This is usually before a meal, indicating I might have waited too long, but I've also had "hypo" symptoms such as racing heart, chills, anxiety and tiredness at times when my BG is in the 4s or 5s.
I don't understand why my BG can dip to 3.2 or 3.8 (also following retesting) with my current diagnosis of pre-diabetic. From what I understand this is rare in non diabetics and DT2.
Is this all down to just being a little late with meals and eating low carb?
Thanks
Nicola
No blinkers. Just trying to stay relevant and on topic. Nor was I arguing the merits or problems of that or similar papers. Just that they were medication based not diet and thus you cannot draw the same conclusions as a result no matter what they are, especially when the report itself points out that the medication is the driving factor behind the detrimental effects seen.You are putting the blinkers on. The paper is not apparently relevant to the OP but may well apply to readers of the thread. Your point is valid, but there is still open ground around it that other papers show similar stories and outcome. The curve is a bathtub curve, and while different medications affect the actual inflection point, there will come a stage where the curve changes slope and rise and going too low seems to be harmful. even in ketosis, our brains and nerves need glucose, which we synthesise from protein if necessary. but that is an inefficient process, and is limited to provide the essential functions for survival. ketosis is actually a form of starvation, and yes, we do appear to be able to sustain a lifestyle with it, but only while there is fat in lipid form to use for overall energy. As I said above, the keto diet is a new kid on the block, and we need to see what happens as it reaches maturity. as an ex-smoker, I am indeed paying the price now in later life.
My twopennoth, i.e.my opinion, people here are encouraged to aim for ketosis and follow a restricted diet (compared to my medium low carb variant) that seems to cause stress in itself. That stress may also be harmful. So I have chosen for myself a less restictive path with a higher (but still technically lowcarb ) approach that reduces that stress and the need to keep to strict diet plan and readings range. The OP is apparently following a keto diet that IMO is not actually necessary in their case, and is showing concern and hints of that associated stress. Pre diabetics should not be advised to go for keto diet IMHO. Low Carb? - yes go for it.
As requested
https://pubmed.ncbi.nlm.nih.gov/23372809/
https://www.healio.com/news/primary...of-lowcarb-lowfat-diet-impacts-mortality-risk
https://jamanetwork.com/journals/jamainternalmedicine/fullarticle/2759134
https://www.bluezones.com/2018/09/n...r-keto-diets-could-lead-to-shorter-lifespan/#
Note the latter link above is from a vegan supporting network and is potentially biassed.
Hi
I got a pre-diabetes diagnosis in December and after reading and researching I decided to alter my eating to low carb.
I test before and after meals and my fasting level.
I've managed to get my readings into the 4s and 5s, but several times a week I catch my BG in the 3s . This is usually before a meal, indicating I might have waited too long, but I've also had "hypo" symptoms such as racing heart, chills, anxiety and tiredness at times when my BG is in the 4s or 5s.
I don't understand why my BG can dip to 3.2 or 3.8 (also following retesting) with my current diagnosis of pre-diabetic. From what I understand this is rare in non diabetics and DT2.
Is this all down to just being a little late with meals and eating low carb?
Thanks
Nicola
Thank you for your replies
My carb intake is very low (less than 22g a day), and yes I'm probably in ketosis. I didn't know I could lower my blood sugar enough to feel ill just by diet alone. :O
I guess that means water fasting is out of the question. I'd heard it can reset all sorts of things including blood glucose problems.
Thank you all for your replies.One thing that may be relevant is that there are recorded events of hypoglycemia occurring post prandial for people who have had gastric interventions (i.e. band or bypass). Apparently this can manifest several years after the operation.
https://www.healio.com/news/endocri...a-a-serious-complication-of-bariatric-surgery
It is probably more associated with the Roux-Y technique, which is the most successful for reversing diabetes.
This makes absolutely no sense to me. Why would you want to maintain diabetic levels deliberately and unnecessarily risk complications?Don't want my body getting used to non diabetic ranges if I am indeed diabetic
aren’t you facing exactly these choices whatever eating style you adopt? Manage it by making particular choices or risk blood glucose and weight gain? That’s not saying you should or shouldn’t choose to continue keto, just that the reality is choices do need to be made. It may be for you low carb without keto strikes the balance suitable for you. And for the record those horrible symptoms may well be temporary anyway not ongoing once you are adapted to the new levels or you find a better balance between carbs and symptoms and levels.I can forsee all sorts of complications including having to continue ultra low carb to avoid weight gain and having to put up with these horrible symptoms, or gaining weight and putting my BG up.
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