Most people with type 2 diabetes will also eventually need to use insulin.

[ringi]

Sorry you got the science a bit wrong. Liver fat is what gives us the very bad insulin resistance.

A low carb diet will prevent an increase in liver fat, and will often results in a slow reduction in liver fat. While also keeping BG within a reasonable range. This is what "eating to your meter" gives people.

A very low carb diet and/or fasting normality result in much faster removal of the fat from the liver, and slow removal of the fat from the pancreas. Hence can give very good BG results. Newcastle diet and 8 week blood suger diet also does the same, but people find it hard to prevent regaining the fat unless they then go onto low carb.

The muscle insulin resistance is not well understood, but is not an issue if we don’t overload our bodies with carbs. Some research shows that muscle insulin resistance can be reduced by resistance training and/or HITT. Muscle insulin resistance seems to also be reduced by long term very low carb diets that have enough protein. But it seems we can never get our muscle insulin resistance down to the level of poeple who are at no risk of Type2.

 

[Oldvatr]

Sorry you got the science a bit wrong. Liver fat is what gives us the very bad insulin resistance.
......

Sorry, but I disagree with what you say here. Here is what the US National Institute of Health has to say about it, and it makes interesting reading,
https://www.niddk.nih.gov/health-in...at-is-diabetes/prediabetes-insulin-resistance
In T2D it is muscle IR that is the dominant feature. Only a few T2D seem to suffer NAFLD, and it is possible to have NAFLD without having diabetes.

 

[ringi]

Look at the research papers that were published by the Newcastle team when they correctly measured the fat in people livers (using their very powerful MRI scanner). Everybody who had Type2 had lots of liver fat, everybody who reversed type2 lost this liver fat. (High BG plus high insulin drives the increase in this fat.)

NAFLD is only detected with blood tests when the liver fat gets to a very high level, but liver fat at a much lower level also greatly increase IR. People can have NAFLD without Type2, as some people can make enough insulin to overcome even a very high level of insulin resistance.

The paper you linked to is about pre-diabetes, and it correctly says that at that point the issue is mostly muscle IR. Muscle IR plus high carbs give us very high insulin, this results in the fat build-up on the liver. But if we remove the high carbs, then the muscle IR is not a problem……

Dr Roy Taylor has proved the “twin cycle hypothesis”, he is not a supporter of low carb, but I think he has correctly discovered the process that goes on when someone reverses Type2.