m_cooper said:i have been really struggling with bg anything from 5.3 up to 15 after meals.this afternoon bg8 went for a 30 min walk and it went down to 6.7 then i had half a packet of salad and 1/2 slice of ham,2 hours after dinner it was 6.3 a miracle for me.how come it has gone down after dinner as i thought it went up
I adore fig rolls a distant memory now though...Romola said:Bye the way - I am feeling a bit naughty because I had a fig roll with my coffee this morning - but only one.
Hana, thats a series of sweeping statements and I don't recognise it at all in the recent dietary advice I've been either been given or read from reputable sources. The best all in one source that I have found on the internet at the moment is the Harvard Nutrition source. It is firmly evidenced based and is well referenced.hanadr said:The medical profession gets fixated on things.
At the moment it's FATS
If you cut fats, which they advise, you need something to take their place. So since Proteins are supposed to injure your kidneys :twisted: (Which is rubbish) the only alternative is carbs. Now even the medics know that sugar leads tto Blood glucose, they advise "Complex" carbs, Ie Starch, which weight for weight turns into double the amount of BG that Table sugar does.
the ideas that don't cross a medical mind very often are: "don't replace sugars and fats. Eat less"
Or "cut the food group that you can't process"
Then there are Food pyramids and the Healthy Eating Plate.
All based on no scientific research and desined to become "Facts"
then they found out that low fat is bad for inants and that some fats are essential to adults too.
Yes, unfortunately that seems to be the case.Doczoc said:Trouble is Phoenix that often GPs and practice nurses are way behind the latest nutritional research. The 'eat plenty of carbs' mantra is still out there and is the advice a lot of newly diagnosed diabetics hear.
Do you suggest I ditch the diet that has overcome all these problems.Insulin resistance isn't just a blood sugar problem. It has also been linked with a variety of other problems, including high blood pressure, high levels of triglycerides, low HDL (good) cholesterol, and excess weight. In fact, it travels with these problems so often that the combination has been given the name metabolic syndrome. (1) Alone and as part of the metabolic syndrome, insulin resistance can lead to type 2 diabetes, heart disease, and possibly some cancers.
Building bone and keeping it strong takes calcium, vitamin D, exercise, and a whole lot more. Dairy products have traditionally been Americans' main source of calcium and, through fortification, vitamin D. But most people need at least 1,000 IU of vitamin D per day, far more than the 100 IU supplied by a glass of fortified milk. (See the multivitamins section, below, for more information on vitamin D needs.) And there are other healthier ways to get calcium than from milk and cheese, which can contain a lot of saturated fat. Three glasses of whole milk, for example, contains as much saturated fat as 13 strips of cooked bacon. If you enjoy dairy foods, try to stick mainly
with no-fat or low-fat products. If you don't like dairy products, taking a vitamin D and calcium supplement offers an easy and inexpensive way to meet your daily vitamin D and calcium needs.
The Survival Advantage of Milk and Dairy Consumption: an Overview of Evidence from Cohort Studies of Vascular Diseases, Diabetes and Cancer.
Conclusions: Set against the proportion of total deaths attributable to the life-threatening diseases in the UK, vascular disease, diabetes and cancer, the results of meta-analyses provide evidence of an overall survival advantage from the consumption of milk and dairy foods.
The results of an initial study of a low carbohydrate diet has surprised many experts who thought that the relative increase in fat content would result in a worsening of glycaemic control and a worsening of lipid profiles rather than completely the opposite. Whilst this work has not changed national guidelines it has made it clear that further work is needed
in this area such as the long-term study funded by Diabetes UK in Exeter. This has also led to the idea that a low carbohydrate diet may indeed be the logical extension of a low glycaemic index diet that has been favoured by many dietitians.
We conclude that weight loss induced by CR favorably alters the secretion and processing of plasma lipoproteins, rendering VLDL, LDL, and HDL particles associated with decreased risk for atherosclerosis and coronary heart disease.
Conclusions Mediterranean and low-carbohydrate diets may be effective alternatives to low-fat diets. The more favorable effects on lipids (with the low-carbohydrate diet) and on glycemic control (with the Mediterranean diet) suggest that personal preferences and metabolic considerations might inform individualized tailoring of dietary interventions. (ClinicalTrials.gov number, NCT00160108 [ClinicalTrials.gov] .)
CONCLUSIONS: These results support the concept that both hyperinsulinemia and a low-fat diet increase DNL, and that DNL contributes to hypertriglyceridemia.
Lichtenstein and Van Horn3 extensively reviewed this approach a few years ago. Examination of their evidence suggests that a low-fat dietary regimen will produce a result opposite to the desired effect: triglyceride levels will actually increase.
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