Now I understand.[Q. UOTE="Squire Fulwood, post: 971199, member: 44622"]
No. I am not saying anything gets diverted. The fat will go normally to subcutaneous cells. Unless the fat had been ingested with an abnormal amount of carbs, then the spiking insulin will store some of the fat in the visceral cells.
The abnormal spike of insulin will be due to the ingestion of simple carbs that will arrive in the blood very quickly, unlike natural complex carbs that will seep to the blood over a longer span of time, and although demanding equal amounts of insulin but this insulin is divided over many hours without peaking.
Hi . My problem with this is that not all obese people are insulin resistant . The figure is about 1in3. We would need an explanation as to why such a large number are not insulin resistantNow I understand.
Maybe the difference is to do with inherited ability to cope with carbohydrates - there seem to be differences about the number of amylase genes people have and apparently different abilities to cope with carbohydrates. I think that's why it's important for each of us to establish our own level of carbohydrate intolerance. Maybe the obese but non insulin resistant have a better ability to cope with dietary carbohydrate but the ability to store an excess of calories as subcutaneous fat?Hi . My problem with this is that not all obese people are insulin resistant . The figure is about 1in3. We would need an explanation as to why such a large number are not insulin resistant
Clive
Hi . My problem with this is that not all obese people are insulin resistant . The figure is about 1in3. We would need an explanation as to why such a large number are not insulin resistant
Clive
Maybe the difference is to do with inherited ability to cope with carbohydrates - there seem to be differences about the number of amylase genes people have and apparently different abilities to cope with carbohydrates. I think that's why it's important for each of us to establish our own level of carbohydrate intolerance. Maybe the obese but non insulin resistant have a better ability to cope with dietary carbohydrate but the ability to store an excess of calories as subcutaneous fat?
A very long video but worth watching, even if just for the first section which discusses the variability of carbohydrate intolerance and insulin resistance:
Information about amylase genes:
Let me use the two tanks example one more time. Imagine two tanks on top of each other and connected to a ground water pump. When the pump is operated what will determine whether the water will start filling the lower tank first or both tanks together is the strength of the pump or the size of the connecting pipes. Now the lower tank is subcutaneous cells, the upper one is visceral cells, the pump is insulin, and the pipes are your genes.
Clive.Hi I understand the basic principal the you are saying but this does not explain why the is 2 pumps working in a different way . You're analogy only works one out of three
Clive
Let me use the two tanks example one more time. Imagine two tanks on top of each other and connected to a ground water pump. When the pump is operated what will determine whether the water will start filling the lower tank first or both tanks together is the strength of the pump or the size of the connecting pipes. Now the lower tank is subcutaneous cells, the upper one is visceral cells, the pump is insulin, and the pipes are your genes.
Hi. If you insist on turning this to physics. GRAVITY is not a variable.
That is easilty explained. People who are obese but not insulin resistant have a favourable fat disposition. The fat is tucked away at non dangerous places, compared to people with type 2 diabetes genetics, they get their fat stored at poor places,liver pancreas and organs.Hi . My problem with this is that not all obese people are insulin resistant . The figure is about 1in3. We would need an explanation as to why such a large number are not insulin resistant
Clive
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