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<blockquote data-quote="Leeannea" data-source="post: 2079017" data-attributes="member: 411549"><p>What you are saying @MeldCP makes perfect sense. I think perhaps the key thing in this article is the paragraph that offers suggestions as to why a low HBA1C increases all cause mortality. The hypotheses focus on hypoglycaemic side effects, effects which nonT1D s don’t encounter, which is why their ideal HBA1C is 5.4. So in theory if a T1D can achieve a low HBA1C without experiencing hypoglycaemia then all should be good. The next logical question is at what levels does this inflammation occur (surely not between 3.5-4.0). I’ve never read of any studies about this. So I think your strategy is great, provided you minimise the frequency and severity of your hypos ( as someone who was eating jelly beans at 4am last night, this can be tricky). Cheers Leeanne</p></blockquote><p></p>
[QUOTE="Leeannea, post: 2079017, member: 411549"] What you are saying @MeldCP makes perfect sense. I think perhaps the key thing in this article is the paragraph that offers suggestions as to why a low HBA1C increases all cause mortality. The hypotheses focus on hypoglycaemic side effects, effects which nonT1D s don’t encounter, which is why their ideal HBA1C is 5.4. So in theory if a T1D can achieve a low HBA1C without experiencing hypoglycaemia then all should be good. The next logical question is at what levels does this inflammation occur (surely not between 3.5-4.0). I’ve never read of any studies about this. So I think your strategy is great, provided you minimise the frequency and severity of your hypos ( as someone who was eating jelly beans at 4am last night, this can be tricky). Cheers Leeanne [/QUOTE]
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