kjc2011
Well-Known Member
- Messages
- 215
- Location
- Salford, Manchester
- Type of diabetes
- Type 2
- Treatment type
- Tablets (oral)
- Dislikes
- Exercising lol
Just an update on my doctors appointment today. He has changed my BP medication so pick my new ones up from the chemist tomorrow. I also discussed I sulin with him. He agrees that it may help, but also said my next HBA1C is due in around 6 weeks, so feels that we should wait and see what the results of these are first as only just started taking the max dose of gliclazide, at the beginning of April, which I can see where he's coming from and makes sense. Thanks for your opinions and sharing g experiences and will keep you updated.
Hi Kristin
As I know you use Lantus as basal insulin. It is a long acting insulin type , but it is impossible to obtain an invariably level all 24 hour. I know that in the lantus and levemir the "product synopsis" postulate that this insulin types are long acting and have a actions time of 24 h, but it is not true.
On the morning the background insulin in the body is low. That is the reason why people with T2DM have a problem on the morning if they digest carbohydrates. This problem can be solved if you take a dose of quick acting insulin type, like Novo Rapid.
The better solution is to change Lantus to Tresiba. Tresiba is a true long acting insulin. It is active for 42 h. I take Tresiba self and I do not experience problems on the morning. I use on the morning Novo Rapid to.
You write that your diet is of low carbo. It is not good if you have T2DM or LADA. If you really have LADA ( it can be verified by test for GAD-65 and C-peptide) you can develop with the time cetoasidosis.
The optimal diet for people with T2DM is to prepare meals with nutrients who have a GI (Glycemik Index) under 50 and GL ( Glycemik Load) under 10.
You must eat carbs for the reason of the biochemical process Krebs Circle and the Biological Respirations Chain . In this process the glucose and the fat are catabolised to CO2 an H2O.
If you do not have carbs in your blood, then this process produces Cottons with negative consequences for you.
I wish to you a good healt
I defer to your greater knowledge of insulin, but I completely disagree with your dietary advice.
I eat low carb, I am T2. I avoid all medication by eating very low carb, I have great energy levels and have no liklihood of ketoacidosis. My blood glucose levels are now normal (below pre-diabetic) and I feel great. I do have ketones, but they are from nutritional ketosis and my body and brain work excellently on them. They are at a level that benefits me, and are no risk of tipping into ketoacidosis.
When I tried eating a low glycaemic index diet I felt dreadful and far less healthy than now.
Hi Brunneria
You have a T2DM. Your pancreas is not affected of the immune system destroying your beta cells.
Kristin write she has LADA. This diagnose is not accepted off the WHO because this disease has not an ICD 10 code.
An expert committee of the ADA with its etiologic diagnostic criteria, has recommended dividing T1DM into type 1A (immune mediated) and type 1B ( other forms of diabetes with severe insulin deficiency). I do not know, but perhaps LADA is the same as T1DM type 1B???
Experts in the Diabetologi, test for LADA with blood analyses for , GAD-65 and C-peptide. If GAD-65 is >20 it indicate for anti bodies for beta cells. Low C-peptide indicate low endogen insulin. The person is going to become T1DM.
I write about ketone to Kristin because she inform that she has LADA.
"You must eat carbs for the reason of the biochemical process Krebs Circle and the Biological Respirations Chain . In this process the glucose and the fat are catabolised in the mitochondria to CO2 an H2O and there is produced ATP". ATP are the molecules who give energy to all biochemical processes. If you have T2DM and you do not eat lot of carbs, then because insulin has defect on the liver to inhibit glucose production , the liver start the gluconeogenese and convert proteins from your muscle to produce glucose. People with T2DM do not become DKA because they have endogen insulin. DKA is a fare more characteristic feature of T1DM than of T2DM, but it may be seen in persons with T2DM under conditions of stress such as occur with serious infections, trauma and cardiovascular or other emergencies.
I do not know how much you know about GI, but you can find it in the Australian site about GI.
Low GI means your digestive tract absorbs the carbs very slowly and the insulin has time to bring them safe on there places. Your blood glucose do not go very high and you can avoid hyperglycaemia.
Have a nice day
PS Remember, the Diabetes has many stage. Your T2DM is on the beginning, but it develops every day. Diabetes is the "Silent killer", and if you do not treat it correct, then the problems will develop.
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