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Type 1 Diabetes
Which is worse? - 1hr at 8mmol or 10min at 15
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<blockquote data-quote="Brunneria" data-source="post: 1082786" data-attributes="member: 41816"><p>Sorry, but that is too simplistic.</p><p></p><p>There are a lot more variables. Size of spike, length of spike, severity of rise and fall.</p><p></p><p>In addition, we would have to distinguish between people who were diabetics-in-waiting, and people who were never-to-be-T2 diabetic, because other variables in their physical makeup could well be predisposing one group to damage, while the other isn't taking damage</p><p>- for instance the diabetics-in-waiting may have abnormal insulin responses for decades before showing significantly raised glucose levels, and raised insulin has been connected to increased coronary risk. </p><p>The resulting complications may be accelerated or even started, before diagnosis, triggered by insulin and glucose fluctuations, but not currently 'visible' using HbA1c and FBG as diagnostic tools.</p><p></p><p>Of course, at the moment, only Kraft's work has shown this (that I know of), but it means that any clear cut observation of spikes is never going to reflect the complex realities.</p><p></p><p>There is also going to be a difference between T1s and T2s, assuming that T1s were 'normal' before contracting T1, whereas T2s could have been brewing complications for years before diagnosis...</p><p></p><p>Having said all of that, studying spike damage has got to be useful, and has got to be worthwhile - it is just very complicated!</p></blockquote><p></p>
[QUOTE="Brunneria, post: 1082786, member: 41816"] Sorry, but that is too simplistic. There are a lot more variables. Size of spike, length of spike, severity of rise and fall. In addition, we would have to distinguish between people who were diabetics-in-waiting, and people who were never-to-be-T2 diabetic, because other variables in their physical makeup could well be predisposing one group to damage, while the other isn't taking damage - for instance the diabetics-in-waiting may have abnormal insulin responses for decades before showing significantly raised glucose levels, and raised insulin has been connected to increased coronary risk. The resulting complications may be accelerated or even started, before diagnosis, triggered by insulin and glucose fluctuations, but not currently 'visible' using HbA1c and FBG as diagnostic tools. Of course, at the moment, only Kraft's work has shown this (that I know of), but it means that any clear cut observation of spikes is never going to reflect the complex realities. There is also going to be a difference between T1s and T2s, assuming that T1s were 'normal' before contracting T1, whereas T2s could have been brewing complications for years before diagnosis... Having said all of that, studying spike damage has got to be useful, and has got to be worthwhile - it is just very complicated! [/QUOTE]
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Type 1 Diabetes
Which is worse? - 1hr at 8mmol or 10min at 15
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