A pilot study to explore the role of a lowcarbohydrate intervention to improve GGT levels and HbA1c
Excess dietary glucose is believed to lead to insulin resistance, type 2 diabetes and also progressive hepatocyte triglyceride accumulation (non-alcoholic fatty liver disease [NAFLD]). Considering the increased cardiovascular risks associated with NAFLD and type 2 diabetes, effective risk factor management of individuals with these conditions is critical. Weight loss can improve abnormal liver biochemistry, the histological progression of NAFLD and diabetes control. However, the most effective diet remains controversial. The authors implemented a low-carbohydrate (CHO) diet in a primary healthcare setting and assessed its effect on weight, HbA1c, serum gamma-glutamyl transferase (GGT) and cholesterol. In this article, the authors present their findings and question the role of dietary CHO in the aetiology of abnormal liver biochemistry and diabetes.
So the research is catching up with my last set of blood tests........