Exactly. T1D have difficulty getting fatty liver. Because the insulin concentration in the liver can never be high enough vs T2D.
It means that T1D, even if they inject huge amount of insulin, they do not get fatty liver... systemic/circulatory insulin concentration cannot never reach the same level as the secreted insulin. Amy Berger's blog explains why this is so:-
http://www.tuitnutrition.com/2019/03/insulin-glucagon-pancreas.html
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But T2D have insulin resistant liver. And chronic hyperinsulinemia via the portal vein will simply mean that it becomes incredibly difficult for T2D to lose liver fats.
So is it logical to eat food that predictably raise insulin levels in the liver if our objective is to lower liver fats? Is a carb lite approach so ineffective and unsustainable in reducing insulin concentration that it should be casually dismissed and ignored?