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Anonymous
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janeecee said:You can only go by the numbers on the meter.
Yes, I get fed up with it all because different people have different risk factors and are at different stages along the continuum when they discover they have a problem. Some people can reverse it if they lose enough weight if that was the cause of the problem and they take action early enough. That doesn't mean they didn't have a problem in the first place.
For me, it isn't a factor and even my modest loss of a few pounds has failed to make any impact. I explained elsewhere my BMI throughout my adult life was consistently around 20, and I wouldn't have maintained that weight on that on a diet of pies and burgers and doughnuts, so there weren't any dietary changes I could make. I've had to drastically cut back on healthy foods like fruit and whole grains because I had nothing else to cut out. I can prevent my BG levels spiking too high after meals but to achieve that I end up on 900-1000 kcal per day, and that's not good. Overall, my fasting is still in the same range as it was 3 months ago but my preprandial levels have increased and my BGs don't fall much between meals. I can't do much more to influence it, so I've come to the conclusion that I've lost beta cells and that I've progressed too far to reverse it.
The fatty liver/fatty pancreas theory of the ND, I can't see it being applicable in my case. No sign of my beta cells "waking up" after being de-fatted. I would like to think otherwise but there's no evidence of it. I think I'd be seeing changes already.
If you can return quickly to low-ish BGs after meals then I don't think the spikes will cause much damage. However, if you remain hyperglycaemic, even at a relatively low level, you can still harm your beta cells as they aren't given the chance to recover between meals. It's a downer for me to know that. I had hoped that something in the ND theory could work for me but I can't see it.
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Today, I had a white bread sandwich (2 slices) with smoked salmon and salad filling and my bGs were 5.0, 9.2 and 4.2 ( at +0, +1 and +2). This peak is consistent with my last OGTT test which was 14.7 at +1:00 after 75g glucose which is about double the carbs of the sandwich. My HbA1C is 38, down from 58 five years ago and my GP, DN and the path lab. all say I am diabetes resolved. In fact, there have been experienced diabetics who are adamant that I never was diabetic in the first place. So both professionals and experienced diabetics are agreeing that my figures are fine for a non-diabetic. Who could argue with that wealth of experience. Therefore, I can only conclude that the above bG pattern is normal for me. So, I ask myself, why be scared of spiking? Isn't spiking perfectly natural? Isn't a spike as a result of glucose secretion into the blood which then triggers insulin secretion (hopefully)?
If I assume that I have been diabetic only since diagnosis (5 years) as I can't go back any further than that, then, if my spiking is bad, would you expect me, by now, to be experiencing complications of one sort or the other and having a higher HbA1C?
Either my GP, DN, path lab and experienced diabetics are all right or they're all wrong since they all agree with each other.