JohnEGreen
Master
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- Nottinghamshire
- Type of diabetes
- Other
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- Diet only
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- Tripe and Onions
When is this bizarre weight regain meant to happen to me? In another 6 months? A year? Two years? I don’t believe it.
He’s still an nhs gp. Do you mean his infographics?Unfortunatley Dr Unwin is no longer used within the NHS
Absolutely agree, I’ll be interested to see. But bear in mind that when raising the carbs I will also reduce the fat to keep overall cals unchanged.When you try your increased carb experiment you may well find out...
I think this is probably at the heart of all this variation in experience and in the diversity of views as to what is cause or effect - namely as you suggest, variations in genes producing a spectrum of metabolic dysfunctions that have been lumped together as “T2D”. I absolutely accept that our mysterious differences have to be handled individually as best we can, on our own mostly given all the confusions and dogmas in the medical profession. But by virtue of those differences I think the view expressed by some that “it’s all just a matter if carbs, carbs, carbs” is not a universal answer to all this. Much more science is needed.And again as my own experiment of one at your stage everything was hunky dory for me. I actually increased calories when I lost weight/got remission because the fat I replaced carbs with had more - but I was able to use the fat ones instead of store them.
It’s not bizarre just because you personally haven’t experienced it - yet. Several of us have told you our experience and you mock us? Or is it just disbelief? Perhaps if your insulin resistance and carb intake carry on the way you are and are planning you’ll do just fine, or maybe you’ll get your own bizarre reaction as it builds up again over time once again.
Honestly I do believe there’s more than one type of type 2. Some respond to simple weight loss really well and that’s all they need to have normal responses. ive seen a number in here go from very high levels to in the 30’s on way way more carbs than me by losing a few stone (like yourself). Whereas I lost lots and cut cubs an awful lot only managed from mid 50’s to low 40’s. Some just don’t respond so well or need much more drastic intervention and I believe there are other, genetic?, issues as play with the likes of me beyond just some visceral fat - which obviously won’t help either.
I should stress that I am not wedded to the Taylor model of T2D causation and remedy, and I do think that team’s research and publications have questionable elements. I would only go as far as saying I think there is much there that is worthy of consideration.
I do take issue with the above comments that the Taylor model expects that maintaining remission long term requires “low calorie” diets, whatever they might be. As far as I can tell, the expectation is only that one should eat a bit less - about three quarters - of what one was eating in the lead-up to developing T2D, in other words to be just a bit more conservative, to eat just enough to fuel one’s energy needs. Take my own case. Before diagnosis I was eating at least 2500 cals daily. To achieve remission I reduced this by about half. Then, during the 18 months since then I reverted to 2000 daily - hardly “low calorie” - and my weight has never risen, nor do I expect it ever to do so. Why on earth should it? When is this bizarre weight regain meant to happen to me? In another 6 months? A year? Two years? I don’t believe it.
He told me exactly the same and so did one of his co-researchers, and so did my special hero Dr David Cavan. We will each find out as time goes on. If it were not true, however, it could only mean that T2D was after all a relentlessly progressive disease despite all we have heard to the contrary from not only Taylor’s team but from wonderful doctors like Cavan, Unwin, Fung and many others. They have all encouraged us to the view that T2D arises from the way we individually store fat through our genes in combination with our dietary behaviour and that it is reversible (for many but not for all) by our behaviour and that it will stay reversed provided we indeed stay trim. Let us hope they are right. I am pretty confident, so far, that they are.In my correspondence with Prof Taylor, I asked him about sustained remission. Paraphrasing, obviously, but his suggestion was provided I remained trim I should be OK.
Post of the thread!AS they say the proof of the pudding is in the eating so eat it and find out Diabetes does not give a hoot for cause and effect or all the discussions or arguments we have it is what it is and not open to discussion.
You can talk all you like about the relative merits of Newtonian or Einsteinian physics and the laws of gravity but when a wall falls on your head you will be injured by it hard hat or not.
Not sure I’d agree that diabetes is not open to discussion! This thread is within a forum category bearing the title “Diabetes Discussions” after all. But I happily acknowledge that there are many who will have no interest in knowing what lies behind the way things operate, and are fully entitled to hold that position.Post of the thread!
Indeed. I reiterate my previous point that the ND diet plan is not just calorie restricted it is also low carb at around 75g / day. So Prof Taylor has plumped for the {weight loss is king} hypothesis, but he has not eliminated low carb as being equally instrumental. It is the elephant in the room. He has not proven his hypothesis. He has not proven that those who lost remission status did so while keeping a low fat pancreas. So his hypothesis remains unproven.Which contradict your hypothesis that remission (or loss of) is weight driven rather than carb led. Maybe I should be introduced to prof Taylor.
On your first point, would you agree that the percentage achieving remission (as they defined it) increased markedly with extent of weight loss? They claimed that nearly 90% achieved it with 15kg but very few with 5kg, as I recall.Indeed. I reiterate my previous point that the ND diet plan is not just calorie restricted it is also low carb at around 75g / day. So Prof Taylor has plumped for the {weight loss is king} hypothesis, but he has not eliminated low carb as being equally instrumental. It is the elephant in the room. He has not proven his hypothesis. He has not proven that those who lost remission status did so while keeping a low fat pancreas. So his hypothesis remains unproven.
The other point to take on board is that studies into the c=peptide test have shown that the more we weigh the more insulin we need. It is a volume effect so BMI is a first approximation. so, sugars will become better controlled as weight drops away, and the control fades as weight goes back on. That is the weight side of the equation. It has nothng to do with remission and it requires a working pancreas.
Weight loss has been a staple dictat for diabetes control for decades as enshrined in the NICE guidelines of first intervention being lifestyle changes only. Taylor has re-invented the wheel in this aspect of ND.
Moreover the ND diet was low fat as well, so would you argue also that he has not eliminated low fat as being instrumental?Indeed. I reiterate my previous point that the ND diet plan is not just calorie restricted it is also low carb at around 75g / day. So Prof Taylor has plumped for the {weight loss is king} hypothesis, but he has not eliminated low carb as being equally instrumental. It is the elephant in the room. He has not proven his hypothesis. He has not proven that those who lost remission status did so while keeping a low fat pancreas. So his hypothesis remains unproven.
The other point to take on board is that studies into the c=peptide test have shown that the more we weigh the more insulin we need. It is a volume effect so BMI is a first approximation. so, sugars will become better controlled as weight drops away, and the control fades as weight goes back on. That is the weight side of the equation. It has nothng to do with remission and it requires a working pancreas.
Weight loss has been a staple dictat for diabetes control for decades as enshrined in the NICE guidelines of first intervention being lifestyle changes only. Taylor has re-invented the wheel in this aspect of ND.
Ah, I can answer that one - low fat is not the key to weight loss. As long as carbs are restricted, fat is fine. Have a look at an old thread of mine.Moreover the ND diet was low fat as well, so would you argue also that he has not eliminated low fat as being instrumental?
Listen. For over 10 years I have successfully controlled my type 2 diabetes only through low carbing (80g a day) and full fat but not extra fats. I have lost NO weight in this time. I can't exercise, for various reasons. My success is ONLY due to my dietary changes.I really don't know why you are having trouble accepting this that this works, for me and others, even if it doesn't work for all or for you. Have you tried low-ish carbing?On your first point, would you agree that the percentage achieving remission (as they defined it) increased markedly with extent of weight loss? They claimed that nearly 90% achieved it with 15kg but very few with 5kg, as I recall.
I think you misunderstand me, tho that may well be my own fault. Through all my T2D journey I have been low(ish) carb or less at 130g, and have been a strong advocate of low carb both here and in other settings. I do not, and never have and never would challenge the validity of others’ methods and experiences. My thread was an enquiry into whether, once remission has been achieved, the constraint upon carbs can be relaxed to some degree without detriment and whether others have tried that. That is all. And I take the view that any objective questioning here is admissible.Listen. For over 10 years I have successfully controlled my type 2 diabetes only through low carbing (80g a day) and full fat but not extra fats. I have lost NO weight in this time. I can't exercise, for various reasons. My success is ONLY due to my dietary changes.I really don't know why you are having trouble accepting this that this works, for me and others, even if it doesn't work for all or for you. Have you tried low-ish carbing?
What, exactly, are you looking for? It is impolite, and impairs objective research, if someone is dismissive the lived experience of others. All real lived experience should be respected and acknowledge. I find your attitude to alternative experiences puzzling.
My point exactly. Cut the weight and the insulin you produce works better. It is not that the pancreas is outputting more, just that what is output has less work to do. Keep the weight off, and it continues to do well. Put weight on and it starts to lose efficiency. As a scientist I was taught to think in terms of Mass not weight.On your first point, would you agree that the percentage achieving remission (as they defined it) increased markedly with extent of weight loss? They claimed that nearly 90% achieved it with 15kg but very few with 5kg, as I recall.
He has notMoreover the ND diet was low fat as well, so would you argue also that he has not eliminated low fat as being instrumental?
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