Yes I totally agree re diet. The carb intake makes insulin balancing a moving target , but that's the official line and it's not budging at the moment sadly. She's not even on fortified drinks / protein shakes .Hi. It sounds like she should be on the Basal/Bolus insulin regime with two different insulins. As a minimum it could be twice-a-day mixed insulin. Having lots of carbs to increase weight is usually the wrong approach as there may not be enough insulin to deal with those carbs with resulting high BS. Having lots of proteins, fats, veg etc is fine.
Eldest son is chasing confirmation from diabetic nurse . He just wants life to be simple and is not querying anything .@stukie ...So, what's the diagnosis - T1/LADA, or T2?
(Sitting on the edge of my seat waiting to hear!)
We were told 5- 8 weeks as it goes to a lab in Cambridge - so hopefully in the right timeframe now. eldest son is acting as carer and is very controlling- he is the point of contact for medics and is quite content to not query anything and keep life simple. We'll see how this week goes.Sitting on the edge of my seat is so uncomfortable, I looked up, 1. the test for LADA (GAD antibody test), and 2.about how long it took to get the results in the UK.
Ten days, it said. Cripes! That's a long time for edge of seat sitting! It's worse than watching a thriller on trad tele rather than binge watching on a streaming program!
But reading my own country's LADA definition, the symptoms do sound like your mother in law's, apart from the emergency DKA? Big weight loss being the big one? (Correct me LADAs! If I have wrongly interpreted info?)
I personally wouldn't do a deep dive into a T1 diagnosis until the results are back... T1 snd T2 really are that different. Including gene/inheritability stats? (I know the T2D ones, am assuming the T1 stats and risk factors are different? Again - LADAs - leap in here....)
There is no official definition of LADA and some doctors chose not to use the term at all.question - is there an inherited susceptibility to LADA given that it has seems to share some aspects of type 1?
Thanks for this info - really interesting re calibration for CGM . I suspect the BS has been set higher to try and assist in weight gain and that this would be a temporary measure until a predetermined amount of weight has been on and then the situation reviewed and BS potentially reduced gradually. As there is deliberately little control on carbs there is a BS rollercoaster. Noone medical seems concerned as weight gain is taking centre stage . I didn't appreciate quite how bad/ uninformed NHS dieticians could be.There is no official definition of LADA and some doctors chose not to use the term at all.
What is agreed is that LADA is a variant of Type 1, not just sharing some aspects of it.
Some define it as Type 1 diagnosed as an adult and some define it as Type 1 with some insulin resistance (a combination of Type 1 and type 2).
Regarding inherited susceptibility, there is a greater chance of getting Type 1 if it is in your family. However, that does not mean descendants will get it just because their parents do and, likewise, it is definitely possible to get Type 1 without any history of it in the family. I was diagnosed in my mid 30s (so, could be defined as LADA) despite no-one else in my family having any type of diabetes.
I am a bit late to this discussion so I apologies for not jumping in and mentioning this earlier.
You mention that your MIL is using a CGM which is great. However, CGMs have limitations. One of these is that they are designed to be most accurate at "normal" BG levels and can be very inaccurate when high or low. Therefore, aiming for a target of 15 with a CGM is like playing darts whilst wearing glasses smeared with vaseline as 15 is considered high (normal BG is between 4 and 7),
I do wonder if the target is 15 as a maximum unless there is a plan to reduce the target: reducing BG too fast can cause additional complications. I recommend confirming this if possible.
Nighttime highs don't suggest anything to do with the liver, it only shows that the amount of insulin active in the body at that time of day is insufficient.Would nighttime highs not suggest a liver issue ?
@AloeSvea @Antje77 ,@EllieM
Thank you .Nighttime highs don't suggest anything to do with the liver, it only shows that the amount of insulin active in the body at that time of day is insufficient.
If I forget to inject in the evening, or if I miscalculate my dose, I'll get high BG.
Thanks @AloeSvea ,Hi @stukie. I feel for you re the blind faith in a diabetes team that your bro in law and mother in law have! Especially re the chain of communication. My own experience with extended family and elderly parents' medical care, and around communication is - there's not much more you can do is breathe deeply in response, and just be there for your loved one! In this case your MIL. Give the info you have gleaned, and breathe deeply again if the eyes of your ill loved one glaze over...
But hey! A diagnosis at last! This is great news indeed. From my own research and maths the percentage of those with both an auto immune diabetes and T2 being treated with insulin is about 25%. My maths may be wrong - always happy to be corrected.
My own understanding and line, re your question a couple of postings above, is that T2D is way more complex than the standard lines about what T2D is, would indicate. And I get the huge focus is on her nutrtional needs right now, rather than how her T2D is playing out? Which is absolutely understandable considering her advanced age.
I know what I do with my own mother and her metabolic health - I do that breathing deeply, and remind myself the focus now, regarding my mother's health, is on her comfort and contentment in her twilight years. I give her support re her big belly, as in - well - don't worry about it and her metabolic health any more! (No ability to change her diet in any case in a rest home.) Give her a lot of ra ra re her reaching such an advanced age. And keep smiling!
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