I've got a theory on this. If a person is T2 (or even predisposed to T2), their pancreas has to work harder to deal with the sugar in their system. The more sugar in the system, the harder the pancreas has to work. Now, if the pancreas is operating at full whack for an extended period of time, it's gonna get worn out, and stop producing insulin altogether.
THAT'S when a T2 will need insulin.
if you have less sugar in your system, your pancreas has a much easier job to do.
P.S - I HATE the thought of treating T2's with insulin. If a person has insulin resistance, the treatment should be to REDUCE the insulin resistance. NOT increase the resistance to insulin by flooding th ebody with high levels of the stuff. Insulin for T2's IS (genarally) BAD. Insulin leads to weight gain, leads to increased insulin resistance, leads to more insulin being injected.
We need to differentiate between T2's. The kind where not enough insulin is produced (but the body is receptive to that insulin - these T2's WOULD benefit from insulin, becasue if they are not resistant to insulin, they are less likelyt o put on weight and head into that cycle of incresed insulin and weight gain), and the kind where insulin is produced, but where the insulin resistance is so high, that the pancreas just keeps on producing it, causing the weight to pile on. The treatment for these 2 sub-categories of T2 are complete opposites.