Only water? Your trigs look maybe a little high..Hi Bulkbiker, yes - I had fasted for 12 hours before the blood test
Hi Bulkbiker, yes - I had fasted for 12 hours before the blood test
Also, I had been doing the HFLC diet since around December if that helps? Initially, I didn't eat enough fat and lost over a stone in weight in a matter of weeks, so I upped my fats intake probably around mid January.
I was enjoying the diet very much and eating absolutely as much as I liked and especially lots of cheese, greek yoghurt, butter, dark chocolate, bacon, treats etc. Despite being very slim I have always had a huge appetite, so we are not talking small amounts here.....! I could see it was controlling glucose and I felt great on the diet....plenty of energy, mentally as sharp as a pin etc. etc. Just the blinking cholesterol that's a fly in the ointment.
Thank you for looking at it with me, Em
Also, I had been doing the HFLC diet since around December if that helps? Initially, I didn't eat enough fat and lost over a stone in weight in a matter of weeks, so I upped my fats intake probably around mid January.
I was enjoying the diet very much and eating absolutely as much as I liked and especially lots of cheese, greek yoghurt, butter, dark chocolate, bacon, treats etc. Despite being very slim I have always had a huge appetite, so we are not talking small amounts here.....! I could see it was controlling glucose and I felt great on the diet....plenty of energy, mentally as sharp as a pin etc. etc. Just the blinking cholesterol that's a fly in the ointment.
Thank you for looking at it with me, Em
Hi,
Hope everyone is as safe and as well as possible.
I was diagnosed with Type 1.5 in Feb. I'm currently still producing some insulin and I'm controlling blood sugars using the High Fat Low Carb diet. However, my last blood tests showed my cholesterol had increased to:
Overall: 6.0
HDL: 1.4
Triglycerides: 1.9
Cholesterol/HDL Ratio: 4.3
Non HDL Cholesterol: 4.6
LDL: 3.7
DN said he was sure this increase was down to diet and asked if I wanted to begin medication. As I wanted to wait for appointment with consultant (in May) I decided to defer that, continue with diet and try to swop in more healthy fats eg. more nuts/avacado/olive oil.....rather than lots of cheese/dark choc/crackling etc. and hopefully keep an eye on what that did to the cholesterol levels - especially good v bad measures.
However, given that it's unlikely I will be able to get a blood test for a long time due to Covid I'll have no idea what's happening. If I don't have plenty of fats I immediately lose weight (I'm 5ft 2 and weigh 7 stone .....I really don't want to lose any weight) so I can't forgo fats - but I'm a bit concerned that I've no idea what's happening with cholesterol.
Has anyone had any similar situation? How did it all pan out? I would welcome any advice/thoughts anyone has - especially before my (presumably telephone appointment) in May. Had my cholesterol been ok then I would have continued with HFLC diet for as long as possible, but now I'm not so sure???
Many thanks, Em.
This is a recent study
A Standard Lipid Panel Is Insufficient for the Care of a Patient on a High-Fat, Low-Carbohydrate Ketogenic Diet
https://www.frontiersin.org/article...NVk2WOgckKNWVK0wligk1arQaAaont2ioRK6VJOpsoOzs
I'll see if I can put it on here.
Abstract from bmjopen.bmj.com
Lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly: a systematic review
Objective It is well known that total cholesterol becomes less of a risk factor or not at all for all-cause and cardiovascular (CV) mortality with increasing age, but as little is known as to whether low-density lipoprotein cholesterol (LDL-C), one component of total cholesterol, is associated with mortality in the elderly, we decided to investigate this issue.
Setting, participants and outcome measures We sought PubMed for cohort studies, where LDL-C had been investigated as a risk factor for all-cause and/or CV mortality in individuals ≥60 years from the general population.
Results We identified 19 cohort studies including 30 cohorts with a total of 68 094 elderly people, where all-cause mortality was recorded in 28 cohorts and CV mortality in 9 cohorts. Inverse association between all-cause mortality and LDL-C was seen in 16 cohorts (in 14 with statistical significance) representing 92% of the number of participants, where this association was recorded. In the rest, no association was found. In two cohorts, CV mortality was highest in the lowest LDL-C quartile and with statistical significance; in seven cohorts, no association was found.
Conclusions High LDL-C is inversely associated with mortality in most people over 60 years. This finding is inconsistent with the cholesterol hypothesis (ie, that cholesterol, particularly LDL-C, is inherently atherogenic). Since elderly people with high LDL-C live as long or longer than those with low LDL-C, our analysis provides reason to question the validity of the cholesterol hypothesis. Moreover, our study provides the rationale for a re-evaluation of guidelines recommending pharmacological reduction of LDL-C in the elderly as a component of cardiovascular disease prevention strategies.
Link to the full paper..
https://bmjopen.bmj.com/content/6/6/e010401.full
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