Greetings all! I was pinged to come here and thought I’d hit a bunch of these questions — hope this helps!
Yes! 16% / 25.7 LDL decrease across the board. I haven’t had a chance to do a more formal write up, but you can see my “tilted ladder graph” here on my LC USA presentation:
Two things:
- Yes — I’d want to keep fasting before the blood test at 12-14 hours, but no more than that. Low quantity or multi-day fasting in particular before a blood test will likely spike your cholesterol numbers.
- Note that if you get to a “healthy weight”, the odds increase that your LDL will go up. My current theory given the evidence is that this is perfectly normal and even expected given your muscle and cardiac tissue require more direct mobilization/delivery from VLDL-originating LDLp (sorry, that was super geeky). Take a look at Lean Mass Hyper-responders to get a sense of it from the far end of the spectrum… cholesterolcode.com/are-you-a-lean-mass-hyper-responder/
Thank you, Guzzler! Indeed, I genuinely believe there’s a serious problem with current studies and how the methodology is determined (especially when done after the data is in to shape a narrative). I’m extremely proud of the fact I announced this hypothesis literally in my presentation before the resulting data was in — which didn’t come until we tested the day afterward.
And yes, I could write about 10k words on why these new patterns completely reset my thinking on current cholesterol studies, but I have a good summary of it near the end of that presentation.
I know mine can. In fact, I just demonstrated this again with my most recent Energy Status Experiment -
http://cholesterolcode.com/energy-status-experiment/
Even if one doesn’t believe in low carb WOE, they should see from my research — at a minimum — that this lipid system is so much more dynamic than we were led to believe.
To be sure, the success rate right now is about 86%, which happens to coincide with the Ketofest experiment. Although, for those 3 where it did rise, it only went up 1-2%, for the 19 that dropped, it fell by 5-38%.
I don't take a formal position on statins as I try to be a good scientist and not claim what I don't know for certain. That said, I have many, many reasons for why I'd likely never go on a statin given what I know now. Even if I were to be convinced high LDL is itself a net all-cause mortality marker (highly unlikely), I'd want to resolve it with diet before a statin... no question.
That’s exactly right! Trigs from your meal are literally being trafficked in your bloodstream via Chylomicrons.
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Hope this was helpful, guys! There’s so much good work on this forum and if I had more bandwidth, I’d be on here quite a bit more often.