xyzzy said:I would like to hear other members thoughts about what has caused the global epidemic in T2 both say at a biological level and wider society based causes.
To get an idea of how prevalent T2 is becoming the UN did an emergency conference on it and the other and other non communicable diseases back last September
Here for the UN communique and links to the recommendations etc.
http://www.un.org/apps/news/story.asp?NewsID=39600&Cr=non+communicable+diseases&Cr1=
As far as I can see the UN is advocating that governments must take much more of a an interventionist stance such as was done in a Chinese study in 1997 that shows you can reverse the increase in T2D if you take an active interventionist approach.
The doc the conference was discussing can be seen here
http://www.un.org/en/ga/ncdmeeting2011/pdf/NCD_draft_political_declaration.pdf
If forum members were "the government" would they be interventionist like the UN suggests? What ways would you intervene?
On a biological level I think my own cause went something like this.
Over a period of years even though I ate a "good" diet I ate marginally too much and gradually put weight on.
As I put weight on I gradually increased my insulin resistance.
As my insulin resistance increased I had to produce more insulin.
The extra insulin produced laid down more fat.
Which made me heavier and my insulin resistance increased..
In the end I could produce no more insulin as therefore my blood glucose levels started to rise.
The rise killed off some of my Beta cells which meant I then produced less insulin but was still insulin resistant.
My blood levels still continued to rise until they went critical and I got T2.
I am now back to the weight I was at around 12 years ago pretty much before my weight began to rise. I am in the high end of the normal range for BMI (24.5) I was diagnosed full T2 with a BMI of (29.2) so I was never officially obese but was overweight.
Want I have no idea about is why I and millions of other people around the world seem to have all done roughly the same thing in the last 40 to 50 years.
For an idea of how DB is spread around the world then this is a good starting position.
Ever tried the IDF's Interactive Diabetes Atlas?
http://www.idf.org/atlasmap/atlasmap
jopar said:So it's a case of turning our perceptions around foods back a bit..
lucylocket61 said:Not all T2's are overweight at diagnosis.
Not all people who are overweight become Diabetic
Not all T2's have eaten ****.
Not all those who eat **** become diabetic
So the cause must be more complex than overeating **** and/or putting on weight.
That is such a sad story about your friend Roy, please accept my condolences. Your last sentence is so true.RoyG said:Maybe it's all of the above predisposition must play a part to a greater extent in the past people may well have been predisposed to getting DB but diet and life styles held it off, now sedentary lives are more pronounced due to all the time savers we have. few people take care of this wonderful machine called our body and it truly is a fantastic piece of Biology, capable of withstanding extremes yet fails sometimes for the simplest of reasons. I had a friend who died last Xmas very suddenly, he had a condition which was called amuloydosis not sure of spelling, it was a disease which laid proteins down on the nervous system and Organs and very rare, he had regular check ups and was classified as disabled and signed off sick, the reason being it could Kill him any second.
Now the twist to the story, I spoke with him on Xmas eve and he was fine and looking forward to his Xmas. Yet two days after Xmas he was found dead in bed, his wife went to work that morning oblivious to the fact he was dead and only found him when she returned 8 hours later. The thing that eventually took him was Swine flue coupled with pneumonia. I have wondered often, how can you be fine one minute then 3 days later end up dead with that?? After all he had gone through. His farther died young from bronchitics suddenly, his sister Died at 37 from renal failure after having cancer of the throat at Ten which went into remission but then developing a drink problem, predisposed hmm a mystery. The truth is the body is a strange mysterious thing, which has mountains left to discover.
lucylocket61 said:Not all T2's are overweight at diagnosis.
Not all people who are overweight become Diabetic
Not all T2's have eaten ****.
Not all those who eat **** become diabetic
So the cause must be more complex than overeating **** and/or putting on weight.
jopar said:They will say, if you want to prevent T1, T2, Cancers and all sorts of things is to fully a Vegan diet as all our ill's are because we ate animal products!
borofergie said:jopar said:They will say, if you want to prevent T1, T2, Cancers and all sorts of things is to fully a Vegan diet as all our ill's are because we ate animal products!
Yeah, we'll try telling that to Robin Gibb.
In conclusion, this study demonstrates that Chinese men and women with type 2 diabetes following an integrated intervention program including diabetes education, frequent blood glucose monitoring and daily use of a diabetes-specific meal replacement, can achieve significant improvements in glycemic control and markers of cardiovascular health.
All study participants were provided diabetes education materials used for monthly group lectures lead by experienced nutritionists from Huadong Hospital. Participants received information on diabetes management, behavioral and lifestyle modification, physical activity, and other diabetes-related health care information, following “Diabetes A-Z,” an American Diabetes Association publication, 3rd edition, (Peking University Medical Center Press, Beijing, China). Additionally, subjects received healthy eating instruction (with sample meal plans) including concepts and applications of food exchanges and lowglycemic foods, based on American Diabetes Association and Chinese Diabetes Association guidelines.
Participants in the Intervention Group also attended weekly sessions at their respective worksites. For each session, lasting approximately 30 minutes, participants met with a research dietitian for diet consultations and a study physician from the company clinic for medical evaluation including assessment of adverse events, a review of blood glucose measurements and adjustment of medications, if necessary. Subjects in the Intervention Group also were provided with blood glucose monitors (Optium TM, Medisense, Abbott Diabetes Care, Alameda, CA) and accessories to encourage frequent testing. To facilitate adherence to the healthy eating plan, participants in the Intervention Group were also provided with a low glycemic, 12 diabetes-specific nutritional meal replacement (Glucerna® SR, Abbott Laboratories, Chicago, IL) that they used to replace breakfast food items such as milk, soymilk, rice soup, or congee at the morning meal. The nutrient profile of the nutritional meal replacement is outlined in Table 1. Cans of powdered formula were provided weekly; participants recorded daily intake on clinical record forms. Compliance to the product intake was assessed weekly by the return of empty formula containers and review of the product intake forms by the study dietitians at each weekly counselling session
xyzzy said:I would like to hear other members thoughts about what has caused the global epidemic in T2 both say at a biological level and wider society based causes.
I had a nice long reply but by the time I went to submit it made me log in again and I lost it (huff!) Shorter version:
The cause of T2 is far from being known. What the UN report and the Diabetes Atlas site both do is back what I already believe - that unhealthy living is the primary cause. Biologically, I think there needs to be huge investment into continuing the research to find out exactly what is triggering insulin resistance. Obesity is one cause of IR and this in turn leads to more weight gain = vicious circle and if untreated = T2 diabetes. However, not all T2 diabetices are overweight. Could visceral fat be playing a part? There is a strong indication that your waist size makes you more liable to develop T2. And what of genetics? Is there a T2 gene? Or are there genetic conditions that predispose some people to weight gain leading to insulin resistance and eventually T2? There's even talk of an immune response where weight gain = excess insulin which triggers a response to kill the insulin. It is hugely complicated but weight does seem to be the most prevalent factor involved.
If I was in charge then a huge investment would be made now to look for the causes and the expenditure would be outweighed by the saving when the reasons are known. I would predict that in 10-20yrs time we will have several different classes of T2 diabetes as the causes become known.
Sciologically, the developing world is producing more economic growth in countries such as India, a move towards urbanisation and Westernisation. This means the fast food culture is reaching all around the globe as is the tendancy to be more inactive and sit infront of one or several screens instead of exercising. People are exercising less, working in less physical jobs and eating worse. This equals weight gain = insulin resistance - etc etc. The developing world is also catching up with the West in terms of life expectancy and as more people live longer, they naturally become less active, gain weight and can develop T2.
The UN's report cites a major need to promote healthy living, diet and exercise and I think our country is doing this already but needs to take it further. There's always going to be exceptions to every rule but healthy living still seems to be the key factor.
I passed a KFC on Saturday and a child with a one year old balloon tied to his chair was getting tucked into fries - the salt intake?? The Olympics are being sponsored by who? Whatever the Government does, it sadly will always come down to human choice and you can't deny people from choosing what's on offer. If I was in charge I would want to intervene and would do wherever possible, especially where you can (ie) in schools. But then they've tried that and people don't like being told what their kids can and can't eat in school. We can only hope to educate people and until we know the exact causes we need to look at the correlations available and use them.
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