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Type 1 Diabetes
Type 1 Diabetes Autoantibodies GAD65 or GAD67
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<blockquote data-quote="Lar oli mu" data-source="post: 1834264"><p>Hi Sam.</p><p></p><p>Datis Kharrazian from Harvard is the one guy along with his team to do real big time research on GAD 65 autoimmune reactivity and it's implications with diabetes. They found out that the structure of the surface of GAD is so similar to the protein structures of several foods, that when they get into the bloodstream the immune system starts mixing them up and starts destroying GAD 65, just because of similar surface structures.</p><p></p><p>The one protein that is causing most of this cross reaction damage is gluten.</p><p></p><p>But there are several other food proteins that cross react with GAD 65. [ATTACH=full]27555[/ATTACH]</p><p></p><p>I reduced my GAD 65 anti-bodies from 120 to 30 just by cutting out the all the cross-reacting foods, that they found in their last study in 2017.</p><p></p><p>My GAD65 antibodies were 120 in 2016 when I fist heard about this playing a role in autoimmune diabetes.</p><p>What this means is: your immune system destroys GAD, which is an enzyme that works in particular in these three areas. The memory center of the brain, the thyroid and the pancreas. In the pancreas GAD has the job to help in GABA production. Without GAD there is no GABA. GABA is the neurotransmitter that sends the signal to the alpha cells to stop producing glucagon and the alpha cells have no stop mechanism.</p><p></p><p>So, if you don't have GAD in your pancreas you have unlimited glucagon. So imagine, when you enter into a stressful situation your Adrenals secrete cortisol and adrenaline which spark a fire in the alpha cells. Glucagon is released and, once in the liver, will result in glycogen being released from the liver, ending up as glucose all over your blood. Unfortunately the alpha cells have no stop mechanism. The stop mechanism is GABA, the neurotransmitter, that you don't have because you don't have GAD anymore. And GAD65 is the most common GAD in the alpha cells.</p><p></p><p>See the disaster with GAD? It is such a small enzyme, but the outcome of this failure is immense</p><p></p><p>Greetings from northern Germany,</p><p>Lars</p></blockquote><p></p>
[QUOTE="Lar oli mu, post: 1834264"] Hi Sam. Datis Kharrazian from Harvard is the one guy along with his team to do real big time research on GAD 65 autoimmune reactivity and it's implications with diabetes. They found out that the structure of the surface of GAD is so similar to the protein structures of several foods, that when they get into the bloodstream the immune system starts mixing them up and starts destroying GAD 65, just because of similar surface structures. The one protein that is causing most of this cross reaction damage is gluten. But there are several other food proteins that cross react with GAD 65. [ATTACH=full]27555[/ATTACH] I reduced my GAD 65 anti-bodies from 120 to 30 just by cutting out the all the cross-reacting foods, that they found in their last study in 2017. My GAD65 antibodies were 120 in 2016 when I fist heard about this playing a role in autoimmune diabetes. What this means is: your immune system destroys GAD, which is an enzyme that works in particular in these three areas. The memory center of the brain, the thyroid and the pancreas. In the pancreas GAD has the job to help in GABA production. Without GAD there is no GABA. GABA is the neurotransmitter that sends the signal to the alpha cells to stop producing glucagon and the alpha cells have no stop mechanism. So, if you don't have GAD in your pancreas you have unlimited glucagon. So imagine, when you enter into a stressful situation your Adrenals secrete cortisol and adrenaline which spark a fire in the alpha cells. Glucagon is released and, once in the liver, will result in glycogen being released from the liver, ending up as glucose all over your blood. Unfortunately the alpha cells have no stop mechanism. The stop mechanism is GABA, the neurotransmitter, that you don't have because you don't have GAD anymore. And GAD65 is the most common GAD in the alpha cells. See the disaster with GAD? It is such a small enzyme, but the outcome of this failure is immense Greetings from northern Germany, Lars [/QUOTE]
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