I am sure there was mention of someone in Dr Atkins New Diet Revolution who had symptoms similar to those you describe - I have a couple of copies of it, different editions, so I am not quite sure of where it was, but the physical need for carbs struck a chord. From memory low carb and supplements were required to stop the symptoms appearing - the standard American diet being deficient in various micronutrients.
Hello - thank you for your reply.
I wanted to mention on different note my average day(s) from a log that I keep...
10/14 - (This was a day I had heavy carbs, but was more lenient than normal)
3:04 AM - 116
6:05 AM - 143
7:08 AM - 175
10:00 AM - 109
1:00 PM - 215
3:03 PM - 183
4:05 PM - 96
8:15 PM - 82
10:00 PM - 86
10/15 -
3:30AM - 46
6:00 AM - 296
7:09 AM - 215
10:00 AM - 92
1:50 PM - 175
3:15 PM - 215
5:50 PM - 111
7:30 PM - 176
8:50 PM - 313
10:00 PM - 210
11:00 PM - 110
10/16 -
3:20 AM - 70
6:00 AM - 134
7:20 AM - 175
9:00 AM - 216
10:10AM - 134
1:10 PM - 52
2:00 PM - 234
4:26 PM - 248
6:43 PM - 143
8:50PM - 175
10:00 PM - 222
11:30 PM - 96
10/17 -
1:46 AM - 122
2:43 AM - 132
6:45 AM - 143
7:45 AM - 175
8:43 AM - 144
10:00 AM - 101
1:00 PM - 86
4:00 PM - 184
5:16 PM - 215
7:09 PM - 202
9:30 PM - 86
And just now @ 90.
Looks like you have a tendency to go high after hypo's. Possibly overtreating?Soooooooooo anyone by chance got any opinions ?
What insulins are you on? Most of us test at the least before meals to decide how much quick acting insulin to take, as it depends on the amount of carbs and on current bg.I don't always check my blood sugar this much but I did so far this week just to see if it was still normal. Prolly won't check it much next week, maybe twice a day next week or something.
Looks like you have a tendency to go high after hypo's. Possibly overtreating?
What insulins are you on? Most of us test at the least before meals to decide how much quick acting insulin to take, as it depends on the amount of carbs and on current bg.
A lot of us test after meals as well to see if a correction is needed. How do you decide on dosing with only 2 tests?
Do you drive? In the UK it's mandatory to test before driving and every 2 hours to prevent hypo's. Even if it's not mandatory where you live I wouldn't dare to drive without knowing where my bg is at.
I'm sorry i can't be of help as to your original question.
Leptin resistance is a delusion. What's actually happening is that insulin is a leptin antagonist. So if you're ODing on insulin, while leptin will still exhibit its effect of cancelling energy conservation peripherally (reproduction will work, etc), it will not cancel hunger in the brain. Cortisol also reduces leptin secretion and leptin response.Research currently into Leptin resistance. Could your Endo check your levels, at least?
What's the problem with protein?Low carb/high fat huh... you know that's an interesting thing right there. Can you give me some examples of low carb/high fat foods? Besides almonds lol. Oh and something that doesn't have much protein.
I wonder why they are spending money on research on leptin resistance when it's a delusion. In the UK.Leptin resistance is a delusion. What's actually happening is that insulin is a leptin antagonist. So if you're ODing on insulin, while leptin will still exhibit its effect of cancelling energy conservation peripherally (reproduction will work, etc), it will not cancel hunger in the brain. Cortisol also reduces leptin secretion and leptin response.
Because they want to do anything except believe that the pathology of hyperadiposity is caused by chronic overdosing of insulin (carb-fat diets, low fat in the susceptible also, cortisol, and fructose abuse) and that alone. Even the "leptin resistance" (in reality, it's another signal that overrides the high leptin and makes you eat even though you aren't hungry) is downstream of the insulin. (OP of course needs to take insulin if they do not want to suffer from a fatal catabolic disease along with high blood sugars - I am not saying insulin is bad, only that overdosing insulin, even in the absence of resulting hypoglycemia, is bad.)I wonder why they are spending money on research on leptin resistance when it's a delusion. In the UK.
Do you have links for these claims @Ellenor2000 ?Because they want to do anything except believe that the pathology of hyperadiposity is caused by chronic overdosing of insulin (carb-fat diets, low fat in the susceptible also, cortisol, and fructose abuse) and that alone. Even the "leptin resistance" (in reality, it's another signal that overrides the high leptin and makes you eat even though you aren't hungry) is downstream of the insulin. (OP of course needs to take insulin if they do not want to suffer from a fatal catabolic disease along with high blood sugars - I am not saying insulin is bad, only that overdosing insulin, even in the absence of resulting hypoglycemia, is bad.)
Obesity research is a "scientific" field full of non-science. They're basically chasing cold fusion up in that thing.
If leptin resistance (as in, a reduction in response to leptin as levels rise independently of any other factor) was real, you would expect fat people to have symptoms of hypothyroidism (since low leptin causes a low triiodothyronine level), indistinguishable from someone who was fat because they actually were hypothyroid. That's not happening.
Elenor, I think reading some of the thyroid fora would demonstrate there are many overweight people exhibiting signs of hypothyroidism with low FT3 levels. One of the issues in U.K. Is the complete resistance of the NHS to routinely test FT3 levels. The NHS is fixated on the useless TSH.Because they want to do anything except believe that the pathology of hyperadiposity is caused by chronic overdosing of insulin (carb-fat diets, low fat in the susceptible also, cortisol, and fructose abuse) and that alone. Even the "leptin resistance" (in reality, it's another signal that overrides the high leptin and makes you eat even though you aren't hungry) is downstream of the insulin. (OP of course needs to take insulin if they do not want to suffer from a fatal catabolic disease along with high blood sugars - I am not saying insulin is bad, only that overdosing insulin, even in the absence of resulting hypoglycemia, is bad.)
Obesity research is a "scientific" field full of non-science. They're basically chasing cold fusion up in that thing.
If leptin resistance (as in, a reduction in response to leptin as levels rise independently of any other factor) was real, you would expect fat people to have symptoms of hypothyroidism (since low leptin causes a low triiodothyronine level), indistinguishable from someone who was fat because they actually were hypothyroid. That's not happening.
I'm a type2 on linsulin therapy and losing weight..... Oh and hv hypothyroidism. I was fighting hunger no matter my diet due to insulin but leptin resistance is a side affect from insulin resistance.... maybe or maybe not.Because they want to do anything except believe that the pathology of hyperadiposity is caused by chronic overdosing of insulin (carb-fat diets, low fat in the susceptible also, cortisol, and fructose abuse) and that alone. Even the "leptin resistance" (in reality, it's another signal that overrides the high leptin and makes you eat even though you aren't hungry) is downstream of the insulin. (OP of course needs to take insulin if they do not want to suffer from a fatal catabolic disease along with high blood sugars - I am not saying insulin is bad, only that overdosing insulin, even in the absence of resulting hypoglycemia, is bad.)
Obesity research is a "scientific" field full of non-science. They're basically chasing cold fusion up in that thing.
If leptin resistance (as in, a reduction in response to leptin as levels rise independently of any other factor) was real, you would expect fat people to have symptoms of hypothyroidism (since low leptin causes a low triiodothyronine level), indistinguishable from someone who was fat because they actually were hypothyroid. That's not happening.
HiYes so both my dietitians (and I've had a few)... as well as endos... the only things they said I could do was use pure will power to stop. They recommended going to a psychologist/psychiatrist and all he could do was prescribe me some anti-depressants as that sometimes is linked to overeating, they say, anyways... therapy didn't help either. I mean I have other mental issues but that isn't linked to my food addiction.
I am almost positive I don't have insulin resistance... my endo said that I don't have that either. based on how much insulin I take and how much I weigh.. I just eat WAY too many carbs.
The reason it gets high is because of the massive amount of carbs I have. Also, the insulin I'm taking is the best for me as the others I've tried actually make my blood sugars slightly worse (because of the reaction time, etc)
Yes I can stop when I feel full, I don't have a problem with that. It's more that I want to get pretty darn full... everytime. *shrugs* I mean if I had the financial ability I would just eat salad most of the time but I don't have enough for that. Anyways, my relationship with food is that I just have to eat a lot... like I said I just do, dunno why... therapist said they think it's just about my will power... with the physical symptoms that I have the therapist got me to a psychiatrist and he prescribed some anti-depressants... many, many other drugs...
Nothing seems to have worked. I would like this to stop but I've been unable to control myself... I've tried home therapies as well...
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