Be wary of this. As a diabetic your reaction to protein and gluconeogenesis (GNG) is not the same as that of a non-diabetic. I've spent a long time looking at this over the past year and have documented my thoughts throughout the forum.I asked this on my weight training forum a while back and the answer given was that you would have to take 300g per day of protein for it to be a problem...
So not a problem for most of us...........
The Keto calculator is good and gave me a range for protein of 110-180 grams per day, depending on how much training I do...
You probably don't need anywhere near what you think. Eat as little as you can get away with, the only way to be sure of netting your requirements is to monitor your lean body mass (not just body weight) and make sure you are either maintaining or gaining (which is very slow unless your taking anabolic steroids).Wow that's what I call a reply! Had to have a shave after reading that
Very interesting tho and it kinda wraps up my life as a very active T1!
T1 is never easy but add in lots of exercise and it becomes even more complicated! So as I exercise a lot I feel that I need more than average qtys of protein to help recovery/muscle growth so I'm confused on what to do? I'm happy with my weight loss and feel that I'm at a suitable weight now. But do I need to be in ketosis to gain all the benefits of using fat for fuel or is there a middle ground where I can eat lchf with higher amounts of protein? AAAAAHHHH!
Also how do I check for protein in urine? Are there test strips available at chemists?
Ta
0.9 ketones with good bg is a healthy level of ketosis for someone on insulin. Don't worry and carry on as you are!I'm a bit confused now cos I'm struggling to get my ketone level above 0.9. I'm only eating 20-30g carbs, I've lowered my protein levels by half thinking this was the problem. How long does it take to get into ketosis and what else could I be doing wrong?
All I know is on LCHF my dietary protein intake raises blood glucose, more protein = higher blood glucose.Glucagon drives gluconeogenesis not the amount of dietary protein. The body should excrete (waste) excess protein.
All I know is on LCHF my dietary protein intake raises blood glucose, more protein = higher blood glucose.
It is the presence of protein that switches on the glucagon secretion in this instance. It does this by triggering an insulin reaction with no carbs present, as insulin is needed for amino acid transport into cells. If you have insulin you need glucose and if this hasn't been eaten, then glucagon is released to get it from the liver processing glycogen. Thus, it is the presence of protein that causes the increase in blood glucose levels.But it's not the presence of protein that switches on gluconeogenisis (or the breakdown of stored glycogen), it's the presence of glucagon. If you can suppress glucagon secreation, the liver will neither make, nor release stored, glucose; not matter how much protein you consume. Conversely, if you stimulate glucagon secreation, the liver will synthesis (from muscle, etc) new glucose, even in the absence of dietary protein.
In a healthy non-diabetic, the beta cells have multiple roles. One is to release insulin and the another is to release Amylin. Amylin slows digestion and crucially suppresses the action of glucagon in the liver. There is also some indication that it can limit the release of glucagon from the alpha cells.So how can I suppress glucagon secretion?
It is the presence of protein that switches on the glucagon secretion. It does this by triggering an insulin reaction with no carbs present, as insulin is needed for amino acid transport into cells. If you have insulin you need glucose and if this hasn't been eaten, then glucagon is released to get it from the liver processing glycogen. Thus, it is the presence of protein that causes the increase in blood glucose levels.
In a healthy non-diabetic, the beta cells have multiple roles. One is to release insulin and the another is to release Amylin. Amylin slows digestion and crucially suppresses the action of glucagon in the liver. There is also some indication that it can limit the release of glucagon from the alpha cells.
As a T1 with no beta cells, you don't have this mechanism. In the US there is a product called Symlin which is synthetic Amylin and it can be prescribed. It is not licensed in the UK due to fears over the hormone causing hypos.
Insulin can also limit glucagon's action, but not terribly efficiently and if you are injecting it, the majority of your insulin hits the muscles and not the liver, where it is needed to reduce GNG.
So how can I suppress glucagon secretion?
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