It doesn't mention type 1 at all though does it? Just says you need have your insulin dose or your oral meds? do Type 1's take oral meds?..unclear at best surely.Did you read all of the first article? It was purely focused on T1 and was pretty reasonable in what it said @bulkbiker
The second article seems to say that if you have ketones you will develop ketoacidosis whatever happens which I'm afraid is also wrong.
It doesn't mention type 1 at all though does it? Just says you need have your insulin dose or your oral meds? do Type 1's take oral meds?..unclear at best surely.
It doesn't mention type 1 at all though does it? Just says you need have your insulin dose..unclear at best surely.
Rereading it, no I guess it isn't clear, although it describes the T1 pizza effect perfectly.
Its a tricky balancing act fats take longer to break down. Fats dont actually increase blood sugar it affects the speed of how carbs can be broken down.
Generally with fatty meals I do 1 injection pre meal and one a few hours afterwards otherwise I have hypos.
This Mendosa article explains what happens once the carbs have stopped effecting bg levels:
So what about after the carbohydrates are finished doing their thing? That’s when the fat itself begins to exert its effects. The process goes something like this:
This is what causes the gradual, delayed blood glucose rise after consumption of large amounts of fat. The response seems to be “dose-dependent” – the more fat you consume, the more insulin resistant the liver becomes, and the more glucose it produces. The type of fat also appears to play a role. Saturated fats (the type found in dairy and animal products) seem to cause more insulin resistance than monounsaturated and polyunsaturated fats (the type found in vegetable products).
- You eat a high-fat meal or snack (this is the fun part).
- In a few hours, the fat begins to digest; this continues for several hours.
- The level of fat in the bloodstream (triglycerides) rises.
- High triglycerides in the bloodstream cause the liver to become resistant to insulin.
- When the liver is insulin resistant, it produces and secretes more glucose than usual.
- The blood glucose rises steadily as the liver’s glucose output goes up.
http://www.mendosa.com/The-Fat-of-the-Matter-How-Dietary-Fat-Effects-Blood-Glucose.htm
He then states they are high in fat.. conveniently forgetting apparently that they are all high in carbs too... As far as I am aware the fats slow the carb impact.. so the carbs don't finish doing their thing they just carry on..
Our findings demonstrate that prediabetic patients show a lower phenotypic flexibility after external aggression, such as OFTT compared with nondiabetic patients. The postprandial response increases progressively according to non-diabetic, prediabetic and type 2 diabetic state and it is higher in patients with liver insulin-resistance.
All I know is eating fat does not raise my blood sugar..as I no longer eat any of the items on his menu I guess I shouldn't comment any further.No I don't think that what it says, read the part where it says Trig levels are temporary raised which in turn leads to insulin resistance, I know as a type 1 eating a high-carb/high- fat and low-carb/high-fat meals my bg levels are raised for several hours after.
Sorry. I meant fats and proteins not carbs.Hi. Both fats and carbs have a small effect on blood sugar and ideally you need to increase the insulin shots a little to take account of them.
Right, but you are not an insulin dependent diabetic. You have functioning beta cells, so the discussion isn't really relevant to you.All I know is eating fat does not raise my blood sugar..as I no longer eat any of the items on his menu I guess I shouldn't comment any further.
charming... I'll butt out then.Right, but you are not an insulin dependent diabetic. You have functioning beta cells, so the discussion isn't really relevant to you.
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