Then it's not relevant to the OP either! I for one found what @noblehead said to be very helpful even though I'm not one of the elite T1s of this forum. If eating fat with carbs increases insulin resistance because of increased trigs even temporarily then it's very relevant to me in my fight to lose weight.
well thought so, do you know you can edit what you already has written but the edit beneath you column in which yo did write the message
I don't normally consider Mendoza as a primary source of info, but in this case he seems to be referring to research that was published last year that seemed to indicate that Type 1's suffer Insulin Resistance as described in the article. Normally IR is condidered to be a mainly T2 thing, and T1's were not generally prone to IR. But this research was on a group of T1's and gives a reasonable hypothesis to explain the pizza effect.
I think there is a basic difference in how IR presents for T1 and T2. In T1 it seems to be mainly the liver that is affected, but in T2 it seems to be muscles that stop using the glucose. Thus T2 meds like Gliclazide work by forcing the pancreas to push out extra insulin that forces the glucose into the cells. Unfortunaltely for T2, this actually forces storage more than burning off in muscles, so T2 tend to expand widthways and put on weight.
I think the answer to that depends on what you do with the triglycerides. If you are converting them to free fatty acids for energy use, then I suspect that it is less of an issue. If you are not using them as energy sources, then I can believe that this would be true.
If you consider that the classic ketogenic diet is not high protein precisely to induce the lipid burning state, it is highly likely that very high proteins will be the cause.
From my experience, excess FFAs seem to be taken out by the gall bladder and excreted via the bile duct. The HF that I think you are referring to here is meant to be part of a ketogenic diet, and so the FFAs are supposed to be burnt off and turned into ketones unstead of using glucose for energy. the large amount od FFAs in the Mendoza article are probably packaged into chylomicrons and VLDL so are not actually free radicals unless the package is damaged during transit (sdLDL). Certainly experimental reports show that higher fat leads to these effects and the blood takes on a milky hue. In this form the body is doing exactly as it should, and it is not (so far) been shown to be dangerous. The liver only releases trigs from storage when glucose levels fall, i.e. ketogenic or starvation or fasting state, and it also needs an absence of insulin to do this..
Not sure about Type 1 but as a Type 2 I have made great improvements with cutting carbs and increasing fat (60% of diet a day). I just now have to watch that I don't over indulge on protein as my doctor informs me that an excessive amount of protein above what the body needs can be turned into sugar which is no good for me at least.
I'll blame my lazy muscles then huh? Is that why I can walk 10 -14 miles at a decent speed and hurt like hell and not lose any weight whilst increasing my BG level then?
What worked for me was only eating within a 10 hour window (between 10am and 8pm as I don't mind missing breakfast). I also alternated between a cooked brunch and light dinner or beef broth/soup brunch and cooked dinner). I guess you have to go with what works and when your body gets used to one thing - surprise it and switch to something else.
I will let another answer this:
