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3 weeks no weight loss

Totto said:
I was advised to put Douglas99 in the Ignore section on the form which I did and I am happy I did so. Douglas was at the same time advised to put me in his ignored section, I have no means to know whether he did so and I don't care, but I did have a peek at what he had written on this thread regarding my post mentioning the book I work on regarding diagnosis and care of hypothyroidism. If Douglas is one of the very few in the UK who gets proper testing for hypothyroidism I would like to hear about how he was diagnosed and what care he has received since.

If Douglas99 don't suffer from hypothyroidism I can not help him. If he is looking for someone who writes books on LCHF I might be able to point him, If he thinks I am writing a book about LCHF he should see someone medical to get him checked for dementia, a far too common side effect of high blood glucose.
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Hmm.
" Douglas was at the same time advised to put me in his ignored section"
do you expect us to really believe you are party to my pm's. or the mods discuss other members of this forum with you?
Cool post on dementia, but you're dealing with adults on here I'm afraid, they'll judge both of us on our posts, as will the mods, who I'm sure will decide on your book advertising.
 
douglas99 said:
Hmm.

do you expect us to really believe you are party to my pm's. or the mods discuss other members of this forum with you?
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Well, no, we didn't go in detail but otherwise yes. Not your personal pm stuff of course.
 
turvell said:
I've created a monster :(


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No,sorry I had to re-ignore for a moment. The monster is elsewhere if anywhere.

My advice is as always: Carbs are bad if you are diabetic. Fats of good quality are always good. To loose weight it is often easier to cut down on carbs compared with cutting calories. Eat to your meter and to satiation. If it works it works, if not, do something else entirely. There is nothing sinister with fat, quite the contrary. It is good for us.
 
I thank everyone for there advice it all helps it really does. I gave up smoking 2 years ago and now I'm tackling my weight and health. I just get down about it sometimes. I've worked so hard for 2 years and just wish I would get thin now


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turvell said:
I thank everyone for there advice it all helps it really does. I gave up smoking 2 years ago and now I'm tackling my weight and health. I just get down about it sometimes. I've worked so hard for 2 years and just wish I would get thin now


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The hardest part is making the effort to lose the weight.
You've done that, so it's easier from now on. Hang on in there, and it will come off.
(By "weight" though, it is fat. If you're gaining muscle, real weight may not change. The mirror can't lie though, so if you like what you see, it's working for you!)
 
Totto said:
And there is nothing sinister about fat.
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Some of the points you make Totto are true but are taken out of context. The points about ketogenesis, insulin release etc need to be read along with all other aspects of how the body digests, uses and stores ingested foods. The passage below is from Taylor's Banting lecture:

"During chronic positive calorie balance, any excess carbohydrate must undergo de novo lipogenesis, and this particularly promotes fat accumulation in the liver. As insulin stimulates de novo lipogenesis, individuals with a degree of insulin resistance (determined by family or lifestyle factors) will accumulate liver fat more readily than others because of the higher plasma insulin levels. The increased liver fat, signalled by rising serum ALT levels in turn will cause relative resistance to insulin suppression of hepatic glucose production. Over many years the resulting hyperinsulinemia will increase further the conversion of excess calories into liver fat. A vicious cycle of hyperinsulinaemia and increased liver fat will become established. Fatty liver leads to increased export of VLDL triacylglycerol, which will increase fat delivery to the islets, with excess fatty acid availability impairing the acute insulin secretion in response to ingested food. Eventually the fatty acid and glucose inhibitory effects on the islets will reach a trigger level, precipitating clinical diabetes. Post-bariatric surgery, the whole mechanism could be thrown into reverse because of profound negative calorie balance. This is a testable hypothesis."
 
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turvell said:
I thank everyone for there advice it all helps it really does. I gave up smoking 2 years ago and now I'm tackling my weight and health. I just get down about it sometimes. I've worked so hard for 2 years and just wish I would get thin now
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Losing weight is easy at the start but does get more and more difficult. But many people also gradually put on weight over many years. It should not be a surprise that after the initial weight loss, which may take a handful of months or even weeks, it takes years to lose the remainder. It is during this time that most people give up and why most diets fail.

That was the whole point of bariatric surgery or the starvation diet used in the Counterpoint Study. The patients had no option.

Try dropping your 1800 per day down to say 1500 per day and increase your exercise by 15 mins per day and see what the effect is in 6 months. Always remember, this is a long term thing. The vast majority of weight loss advice is aimed at people who want to look good in a bikini on the beach next summer. The changes you make have to be both beneficial for your health and sustainable for the rest of your life. You have to be comfortable with them. It is worth the investment in the time and effort taken in learning what these changes are.
 
Yorksman said:
Some of the points you make Totto are true but are taken out of context. The points about ketogenesis, insulin release etc need to be read along with all other aspects of how the body digests, uses and stores ingested foods. The passage below is from Taylor's Banting lecture:

"During chronic positive calorie balance, any excess carbohydrate must undergo de novo lipogenesis, and this particularly promotes fat accumulation in the liver. As insulin stimulates de novo lipogenesis, individuals with a degree of insulin resistance (determined by family or lifestyle factors) will accumulate liver fat more readily than others because of the higher plasma insulin levels. The increased liver fat, signalled by rising serum ALT levels in turn will cause relative resistance to insulin suppression of hepatic glucose production. Over many years the resulting hyperinsulinemia will increase further the conversion of excess calories into liver fat. A vicious cycle of hyperinsulinaemia and increased liver fat will become established. Fatty liver leads to increased export of VLDL triacylglycerol, which will increase fat delivery to the islets, with excess fatty acid availability impairing the acute insulin secretion in response to ingested food. Eventually the fatty acid and glucose inhibitory effects on the islets will reach a trigger level, precipitating clinical diabetes. Post-bariatric surgery, the whole mechanism could be thrown into reverse because of profound negative calorie balance. This is a testable hypothesis."
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Happy so long as something is right. I do however fail to understand what your post on fatty liver has to do with dietary fat. Or do you agree with me that dietary fat and a low carb diet will help a fatty liver recover?
 
Oh Hun I know how you feel I can go six weeks and not loose an ounce but don't worry it will start to go, but I find is I drink plenty of water and high fibre food it comes of easier , I do at least 30mins of exercise a day which dose help the glucose too and try to do a lot of stretching it helps the muscles .... My physio therapist told me that and it works too hope I'm off help xxxxx


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Totto said:
I do however fail to understand what your post on fatty liver has to do with dietary fat.
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There is a direct link. Saturated fatty acids from animal products, trans fatty acids in meat and milk resulting from bacterial biohydrogenation of unsaturated fatty acids in the rumen and partial hydrogenation of unsaturated fatty acids in vegetable oils which occur in the processing can all lead to lipotoxicity in the liver as well as several other organs, (Lipotoxicity: effects of dietary saturated and transfatty acids). Lipotoxicity is a key factor for the progression of fatty liver disease by inducing hepatocellular death, activating Kupffer cells and an inflammatory response, impairing hepatic insulin signalling resulting in insulin resistance, and activation of a fibrogenic response in hepatic stellate cells that can ultimately lead to cirrhosis, (Fatty liver and lipotoxicity)

Type 2 diabetics all have an excess of liver fats as it is and although ketogenic diets can result in weight loss elsewhere and although the severe restriction on ingested carbohydrates results in low plasma glucose levels, there is an increased risk to the liver as well as other organs, especially the kidney. Whilst fat is an important component in human nutrition, as with any food type, exclusivity or excess in the long term is to be avoided. As with many situations, it is unwise to sacrifce the long term in favour of the short term, however tempting the latter may be.
 
http://www.ncbi.nlm.nih.gov/pubmed/24115747

"CONCLUSIONS:
These data support the use of a short-term low-carbohydrate diet whenever a reduction in liver volume is desirable. Overeating carbohydrate is harmful because it increases liver volume."

http://www.diabetesincontrol.com/in...ntent&view=article&id=10796&catid=1&Itemid=17

And a study on the fact that full-fat milk helps us to stay lean, compared with low-fat. http://www.ncbi.nlm.nih.gov/pubmed/23320900
and another on the same subject: http://www.ncbi.nlm.nih.gov/pubmed/22810464:

CONCLUSIONS:
The observational evidence does not support the hypothesis that dairy fat or high-fat dairy foods contribute to obesity or cardiometabolic risk, and suggests that high-fat dairy consumption within typical dietary patterns is inversely associated with obesity risk. Although not conclusive, these findings may provide a rationale for future research into the bioactive properties of dairy fat and the impact of bovine feeding practices on the health effects of dairy fat.
 
Totto said:
http://www.ncbi.nlm.nih.gov/pubmed/24115747

"CONCLUSIONS:
These data support the use of a short-term low-carbohydrate diet whenever a reduction in liver volume is desirable. Overeating carbohydrate is harmful because it increases liver volume."

http://www.diabetesincontrol.com/in...ntent&view=article&id=10796&catid=1&Itemid=17
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This study points the finger at high carb and low fat as the culprit in NAFLD

Metabolic syndrome is associated with greater histologic severity, higher carbohydrate, and lower fat diet in patients with NAFLD.

http://www.ncbi.nlm.nih.gov/pubmed/17032189
 
Totto said:
http://www.ncbi.nlm.nih.gov/pubmed/24115747

"CONCLUSIONS:
These data support the use of a short-term low-carbohydrate diet whenever a reduction in liver volume is desirable. Overeating carbohydrate is harmful because it increases liver volume."
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Demonstrating a short term benefit of a low carbohydrate diet over a low calorie diet has nothing to do with the papers you asked for which address the long term dangers of a high fat diet.

You stated that you "fail to understand what your post on fatty liver has to do with dietary fat" and I cited two paper which explain what the connection is. It is a long term danger. You may disagree but the papers you cite do not say what you think they say.
 
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I am sorry Yorksman, I tried to read the paper links you posted but have to admit I lack the energy to go through them thoroughly enough to understand them properly. Granted, industrial trans fat should be banned. And most vegetable oils trigger inflammatory processes and should be avoided. Let´s see what other new research we can find. I think I will start a few threads on cons and pros.

As to diary fat and other animal fats I am not so sure. As you might have seen in some of my links recent research indicates they are good for us

The research I have been able to understand states that the real culprit in non-alcoholic fatty liver disease is carbs.

By the way, is it really true all t2 have fatty liver? How do you go about the diagnosis of fatty liver? Can you have fatty liver and normal liver enzymes? Will it show on ultrasound? Or do you have to have a biopsy?
 
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