Thanks for this noblehead. Although I am type 2 it's of interest to me because I am insulin resistant and I'm also a bit inquisitive by nature so I like reading what type 1's say too.
Last edited by a moderator:
Thanks for this noblehead. Although I am type 2 it's of interest to me because I am insulin resistant and I'm also a bit inquisitive by nature so I like reading what type 1's say too.The following member( who is type 1 and follows a LCHF diet) found that their insulin usage went down when they changed the type of fat in their diet:
http://www.diabetes.co.uk/forum/threads/saturated-fats.48599/#p438010
So in summary raised blood lipids cause temporary higher basal requirements?I'm not too sure, however this is how Scheiner explains how a high fat meals causes insulin resistance:
So what about after the carbohydrates are finished doing their thing? That's when the fat itself begins to exert its effects. The process goes something like this:
This is what causes the gradual, delayed blood glucose rise after consumption of large amounts of fat. The response seems to be dose-dependent the more fat you consume, the more insulin resistant the liver becomes, and the more glucose it produces. The type of fat also appears to play a role. Saturated fats (the type found in dairy and animal products) seem to cause more insulin resistance than monounsaturated and polyunsaturated fats (the type found in vegetable products).
- You eat a high-fat meal or snack (this is the fun part).
- In a few hours, the fat begins to digest; this continues for several hours.
- The level of fat in the bloodstream (triglycerides) rises.
- High triglycerides in the bloodstream cause the liver to become resistant to insulin.
- When the liver is insulin resistant, it produces and secretes more glucose than usual.
- The blood glucose rises steadily as the livers glucose output goes up.
But we have been here before with this discussion Dillinger, didn't your consultant think that your insulin resistance was down to your LCHF diet?
Or alternatively you could describe it as a mini liver dump triggered by dietary fat ?So in summary raised blood lipids cause temporary higher basal requirements?
Sent from the Diabetes Forum App
Many thanks noblehead, this is really interesting. I'm trying to get my head around it.
- High triglycerides in the bloodstream cause the liver to become resistant to insulin.
- When the liver is insulin resistant, it produces and secretes more glucose than usual.
Looks like they used an 18 hour glucose clamp so it's a real increase in insulin requirement and not just a time shift. But yes it's the "pizza effect" of fat plus carbs. I don't ever get this effect from fat on LCHF. I hate how these studies just take high carb as a given rather than controlling it properly as a variable. It's scientifically sophomoric.
High blood lipids cause immediate but transient "insulin resistance" in non diabetics and T2 by suppressing beta cell activity. I wonder what the mechanism is in T1?
Scary that Joslin are using this explicitly to argue for low fat diets when it actually argues more strongly for a low carb diet.
I wish they would give a link to the actual paper but from the write up the study was not isocaloric ?
Sent from the Diabetes Forum App
Thanks for this noblehead. Although I am type 2 it's of interest to me because I am insulin resistant and I'm also a bit inquisitive by nature so I like reading what type 1's say too.
personally i think its true as your body uses insulin to break down carbs ect then when your type one u no longer have the natural ability to break down the carbs ect so you body is technically unable to tolerate carbs without injectionSaw a post on the DCUK facebook page today, and thought it was quite interesting:
I just wanted to ask what people think of Carol - I think she's right... although I had a smile when I read it!
personally i think its true as your body uses insulin to break down carbs ect then when your type one u no longer have the natural ability to break down the carbs ect so you body is technically unable to tolerate carbs without injection
personally i think its true as your body uses insulin to break down carbs ect then when your type one u no longer have the natural ability to break down the carbs ect so you body is technically unable to tolerate carbs without injection
If the body couldn't break down the carbs we wouldn't need to insulate for them. Its the fact our bodies can break them down that raises our levels.
Hiding now
Type 1, pumping nova rapid
I did not vote at all because I have no Idea. I feel as if I am glucose intolerant.PS. I'd love to know who abstained and why!x
Er the topic of the thread isn't "Can diabetics *digest* carbs ?"If the body couldn't break down the carbs we wouldn't need to insulate for them. Its the fact our bodies can break them down that raises our levels.
Hiding now
Type 1, pumping nova rapid
I'm one of two abstainers I think and we both posted explaining our reason why, which is we don't agree with the word "just" in the topic question.PS. I'd love to know who abstained and why!x
It is not cant they either.
Correct. It's not about digestion of carbs. It's not even strictly about whether we can utilise carbs. It's about whether ingesting carbs has adverse effects.It is not cant they either.
We use cookies and similar technologies for the following purposes:
Do you accept cookies and these technologies?
We use cookies and similar technologies for the following purposes:
Do you accept cookies and these technologies?