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Sir Steve Redgrave and treatment decisions

Yes I agree. The ignorance displayed by some on this site is equally shocking.
Yes. That's why site is useful. We all need some level of education. I for sure don't know everything. If I did I'd be very rich. Just the opposite. :(
 
I beg your pardon??? where do you get off coming out with such an insulting comment? Have you learned nothing about the many causes and symptoms of t2 during your time on these boards?

I respectfully suggest you re-think your comment.

(and before someone flags this as insulting or a personal attack, I am responding to an insult to all of us who are overweight t2's)

I respectfully confirm that I absolutely will not rethink my comment.

I was (as others have suggested) merely reflecting the oft stated "red top newspaper" view of T2 diabetics which seems to be sadly prevalent in the general population. Which is easily refuted by using Sir Steve as an obvious example.

If you feel personally insulted (on behalf of others) then that of course is your right.
 
Yes, I have wondered how rich and famous T2 diabetics have been doing with treatment choice and support and so on more than once
Well Stephen Furst died of complications a couple of weeks ago so maybe being rich and famous doesn't help that much.

Among his many accomplishments he was author of a book titled "Confessions of a Couch Potato."
 
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I respectfully confirm that I absolutely will not rethink my comment.

I was (as others have suggested) merely reflecting the oft stated "red top newspaper" view of T2 diabetics which seems to be sadly prevalent in the general population. Which is easily refuted by using Sir Steve as an obvious example.

If you feel personally insulted (on behalf of others) then that of course is your right.
I read the line as a tongue-in-cheek dig at the Daily Mail type stereotyping. It can sometimes be difficult to convey sarcasm or irony or humour in pixels, but in the context of your posting history, I was sure you didn't mean anything bad.
 
There is another view for athletes, to become fat adapted. See ...
 
I wonder what his c-peptide test result would be? He may even be a Late onset T1 and not T2, although I would hope his consultant has carried out the test and not just guessed T2.

Yes, this had me wondering as well.

At the moment I am assuming that he was correctly diagnosed as T2 but the only available treatment option which also allowed him to maintain his peak performance on a mainly carbohydrate diet was insulin.

It would be good to know if he was able to modify his approach once he quit the intense training regime.

However I do know people (including one of my cycling group) who decided they would rather maintain their current eating style (which may or may not have contributed to their diabetes) with insulin than try a major lifestyle change.

IMHO a valid lifestyle choice which I would not make.

One illustration of the downside of this approach is the inherent hypo risk. My cycling buddy is also a dingy sailor who had to give up single handing in case of a hypo when out on his own. There are also implications for driving.

I am also needle phobic, which is an added incentive to stay off insulin.
 
Yes, this had me wondering as well.

Wonder no more. Here is a paper written by Sir Steve, his wife (a doctor) and his diabetes specialist. I think it's fair to say that with his physical health being closely monitored because of his sporting abilities, he didn't have to go to his GP.

http://www.clinmed.rcpjournal.org/content/3/4/333.long

I found that it had been discussed 3 years ago on this forum. Interesting paper, hopefully containing the answers to any questions you might have.
 
Wow! A really great revision text for glucose metabolism in performance athletes.

I need to go back to Volek and Phinney to review how anaerobic muscle metabolism works for the fat adapted.

There is also the hint that extra insulin immediately post exercise may assist the non-diabetic in hepatic carb loading after exercise.

TL;DR the big problem identified was reduced production of glucose by the liver during intense exercise (due to reduced uptake post exercise). Solved by loading more carbohydrates into the liver immediately post exercise.
 
Wonder no more. Here is a paper written by Sir Steve, his wife (a doctor) and his diabetes specialist. I think it's fair to say that with his physical health being closely monitored because of his sporting abilities, he didn't have to go to his GP.

http://www.clinmed.rcpjournal.org/content/3/4/333.long

I found that it had been discussed 3 years ago on this forum. Interesting paper, hopefully containing the answers to any questions you might have.

Mr. Picky, having read the report, notes that the initial diagnosis was through his GP which is the correct way to do things. Even GPs have GPs.

However all the subsequent heavy lifting was done by a specialist team.
 
having read the report, notes that the initial diagnosis was through his GP

I should have said "he didn't have to go to his GP for ongoing treatment" and maybe have added "like what we does" LOL I think I read somewhere that he has a pump, probably not available through normal channels. Good luck to him, having literally bumped into him (I should have noticed the medal) I feel as though I know him. At the time I'd actually been playing tennis (badly) and who should walk on as we walked off but Tim Brook Taylor. I mix in such high circles you know LOLOLOLOLOL
 
I wish that people with no insulin resistance and who diabetes results from the side effect of drugs were not called “Type2”, as they seem a lot closer to a “part Type1”……

There is too much confusion by putting everyone who is not a clear case of Type1 in the Type2 box.
 
I seem to remember it being referred to as type 3E but that no longer seems to be the case my diabetic consultant got quite shirty when I referred to my self as type 2 saying you have drug induced not T2 diabetes. Makes no nevermind to me though.
 
It makes a big difference as people with recent "true type2" can often turn it round just by losing weight and be "back to normal".
 
It makes a big difference as people with recent "true type2" can often turn it round just by losing weight and be "back to normal".
If only losing weight was simple for all of us.... :(
 
One thing they don't seem to test for.
If you have a fully functioning pancreas on diagnosis then there is a chance for complete reversal.
If your pancreas is already under performing then a different strategy may be optimal.
 
If only losing weight was simple for all of us.... :(

If it was, then we would not have insulin resistance and hence Type2 with some sort of reduce carb being the only workable way to lose weight.
 
One thing they don't seem to test for.
If you have a fully functioning pancreas on diagnosis then there is a chance for complete reversal.
If your pancreas is already under performing then a different strategy may be optimal.

It has been shown that a "pancreas is already under performing" can sometimes be turn around by removing the fat from round it, also if insulin resistance is reduce enough then a 100% working pancreas is not needed. We all also know that Low Carb lets a part working pancreas produce test results that look normal.
 
It makes a big difference as people with recent "true type2" can often turn it round just by losing weight and be "back to normal".
It took some drastic action and over a year but I am now in remission so it can be done even if you are drug induced.

Mind you how long that will last I have no idea, if the steroids nock me out of remission then game over I think.
 
Wonder no more. Here is a paper written by Sir Steve, his wife (a doctor) and his diabetes specialist. I think it's fair to say that with his physical health being closely monitored because of his sporting abilities, he didn't have to go to his GP.

http://www.clinmed.rcpjournal.org/content/3/4/333.long

I found that it had been discussed 3 years ago on this forum. Interesting paper, hopefully containing the answers to any questions you might have.
Interesting and I note the phrase 'detectable c-peptide'. I suspect many T1s have detectable c-peptide as well but the key is where it is in the accepted range; we will never know.
 
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