Hi all. Long story but I maybe a type 2 from a type 1..and I'm having to think about trying to improve my weight and sugar levels drastically. I have recently started metformin and I'm on Fiasp and Levemier.
I've tried all kinds of diets and not been successful at one. I've done sw and ended up eating too much, starved at ww and felt sick on lchf.
Willing to try anything now as I've just realised I'm eating whatever I want and injecting insulin for it yet my levels are sky high.
Any advice welcomed xx
YouMUST eat carbs because your body need the energy molecule ATP (Adenosine Triphosphate).
sorry but no, not correct. There have been several T1's on here who take metformin for many years.If You use metformin then you have DM T2 and not DM T1.
This is factually incorrect. The liver can endogenously manufacture all of the body’s glucose requirements. Additionally, mitochondria can also use ketone bodies as well as glucose to synthesise ATP.
Is incorrect that Your body need ATP for all biochemical process?
The hepar (Liver) do not generate ATP, but mitochondrial process produces ATP in the respiratory chain.
It is not possible to write on the little place all about this very complicated problem.
You can correct me for all wrong spelled words and sentence syntax. IT IS OK from me.
Thank You
No. It’s just incorrect that you need to consume exogenous carbohydrate in order for any of this complex process to take place. The liver does not need to manufacture ATP. ATP is made inside the cell from glucose or ketones passing through the cell membrane. The body will normally maintain glucose homeostasis within a very narrow corridor, irrespective of whether or not the individual consumes glucose. Therefore the rationale that one must consume carbohydrate in order to supply ATP is erroneous. One can of course choose to consume it if they wish, but that is another matter.
Why would the liver take amino acids from protein (usually ingested rather than catabolise that already in the body) when there is already an excess of glucose? This is now thought to be a demand driven process rather than as you describe.Insulin “collect “glucose in the liver as glycogen and glucagon release the glucose from the glycogen and the liver moreover make GLUCONEOGENESE, i.e. it takes some amino acids from the skeletal muscle and convert them to glucose.
The risk by DM T1 is the person can get KETOACIDOSE and the consequence of that is DED.
Not so. Diabetic ketoacidosis happens in the presence of three factors - extremely high blood glucose, total lack of insulin and dehydration. Ketosis is the burning of fat for fuel. One does not lead to the other.
Mitochondria do not use ketone bodies, but mitochondria produce ketone bodies if your body catabolizes FAT, because you do not have glucose.
The risk by DM T1 is the person can get KETOACIDOSE and the consequence of that is DED.
To Mel dCP
Very simply explained, Ketoacidosis mean Your blood has to much acid from the ketone.
The human blood has property as buffer. But there is a limit how much acid can accumulate in the blood.
Ketoacidosis is a condition where in the blood is much more acid then the buffer can accumulate. The consequences of that are fatale and the person die of this acid.
I know exactly what it is, as an ex research biochemist, science teacher, trainee medic and a type one of 20 years who has suffered from it (and survived) twice in the past - due to having the three necessary requirements for DKA - namely extreme hyperglycaemia, no insulin and severe dehydration. I have eaten a very low carb, ketogenic diet for over 18 months now, and I’m still not dead. Sorry about that.To Mel dCP
Very simply explained, Ketoacidosis mean Your blood has to much acid from the ketone.
The human blood has property as buffer. But there is a limit how much acid can accumulate in the blood.
Ketoacidosis is a condition where in the blood is much more acid then the buffer can accumulate. The consequences of that are fatale and the person die of this acid.
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