chiefsub68
Member
- Messages
- 22
- Location
- Colchester UK
- Type of diabetes
- Type 2
- Treatment type
- Tablets (oral)
- Dislikes
- Dentists.
It depends on how well diabetes is controlled. - i know people who are T1/T2 with complications - amputee or blind and on dialysis because of bad control... looking at your hba1c.. it isn't bad you can improve my last few have been in the 80s and mid- 70s. and my most recent hba1c was 76. i have a few problems which i am just living with feet and eyes but my main focus is to keep my levels down.. the higher and longer the blood sugar is up the more damage it will do.Hello, I was 53 when my T2 was diagnosed nine years ago. Since then my HbA1C has gone up and down – 45, 43, 41, 46,45, 51, 52, 57 (ouch), 50, 68 (ouch) and most recently back to 58. I initially controlled my diabetes by diet and exercise but I am now on 2 x Metformin, diet and exercise.
My question is this: when do people's harsher symptoms kick in, and do they do so comparatively quickly? So far, touch wood, all my sight, feet, and kidney tests have been fine. Thanks in advance. Will
The good news, is it can be slowed down.Hello, I was 53 when my T2 was diagnosed nine years ago. Since then my HbA1C has gone up and down – 45, 43, 41, 46,45, 51, 52, 57 (ouch), 50, 68 (ouch) and most recently back to 58. I initially controlled my diabetes by diet and exercise but I am now on 2 x Metformin, diet and exercise.
My question is this: when do people's harsher symptoms kick in, and do they do so comparatively quickly? So far, touch wood, all my sight, feet, and kidney tests have been fine. Thanks in advance. Will
Hi, not sure there's a simple answer. For example, one of the reasons the medics chose an HbA1c level of 48 as the point where they would diagnose diabetes is that retinopathy is "rare" - ie not unknown - at levels lower than that. My experienece was to have a range of symptoms while my A1c was in the low 40s and rising: I was told on several occsions that I wasn't diabetic, despite the symptoms, because I hadn't reached the magic 48. I also know people who have much higher HbA1cs than I ever had and have had exactly no symptoms.Hello, I was 53 when my T2 was diagnosed nine years ago. Since then my HbA1C has gone up and down – 45, 43, 41, 46,45, 51, 52, 57 (ouch), 50, 68 (ouch) and most recently back to 58. I initially controlled my diabetes by diet and exercise but I am now on 2 x Metformin, diet and exercise.
My question is this: when do people's harsher symptoms kick in, and do they do so comparatively quickly? So far, touch wood, all my sight, feet, and kidney tests have been fine. Thanks in advance. Will
Hi Chris. My weight has stayed essentially the same. When I was first diagnosed I went on a powder diet and lost a lot of weight but (as always happens with sudden weight loss) it crept back on within seven months. I'm going to cast around to see if I can get rid of the more carby parts of my diet.Hello Will, has the gradual rise in A1c been accompanied by a gradual gain in weight or waist size also, or have they remained essentially the same?
My understanding is that if one’s initial weight loss has been sufficient to clear excess fat from the pancreas and so get one’s A1c down into the remission zone then allowing weight to rise by as little as 6 lbs can be sufficient to put one back into non-remission. It’s that thought that keeps me glued to the bathroom scales week after week after week, probably for ever.Hi Chris. My weight has stayed essentially the same. When I was first diagnosed I went on a powder diet and lost a lot of weight but (as always happens with sudden weight loss) it crept back on within seven months. I'm going to cast around to see if I can get rid of the more carby parts of my diet.
That's really interesting. I was only diagnosed in July with an a1c of 116 (eek!) but was really strict on the low carb, lost about 14lb (not over weight for the 1st time ever!) and 3 months later having stopped the metformin about 1 month in, my a1c was 42 and my fasting finger prick tests were coming in under 6mmol/l.My understanding is that if one’s initial weight loss has been sufficient to clear excess fat from the pancreas and so get one’s A1c down into the remission zone then allowing weight to rise by as little as 6 lbs can be sufficient to put one back into non-remission. It’s that thought that keeps me glued to the bathroom scales week after week after week, probably for ever.
I believe it takes only half a gram of fat in the pancreas to compromise the insulin signalling or secretion, so if one’s (dratted!) genes are disposed to direct excess fat to the pancreas rather than subcutaneously then that’s how easy it is for a little weight gain to undo our remission. My concern is that there may be people who have not expelled that fat but whose bg is kept low by low carbs alone. And although the latter situation is a good thing while it lasts, I have seen reports that long term that fat may damage beta cells permanently and take away the diet-only option of good T2 management. So I believe that carb reduction has a powerful role to play but that the fat stripping is a greater priority and is not getting enough emphasis.That's really interesting. I was only diagnosed in July with an a1c of 116 (eek!) but was really strict on the low carb, lost about 14lb (not over weight for the 1st time ever!) and 3 months later having stopped the metformin about 1 month in, my a1c was 42 and my fasting finger prick tests were coming in under 6mmol/l.
Unfortunately I slacked off a bit and the weight crept back on and my fasting finger prick levels went up over 7. As I've lost the weight again, the finger prick numbers are slowly coming down again. I'm hoping that if I get to the lower weight again (and a bit more to be honest) and actually stay there this time, I can officially go in to remission.
And the best way to fat strip remains lower carbs and exerciseI believe it takes only half a gram of fat in the pancreas to compromise the insulin signalling or secretion, so if one’s (dratted!) genes are disposed to direct excess fat to the pancreas rather than subcutaneously then that’s how easy it is for a little weight gain to undo our remission. My concern is that there may be people who have not expelled that fat but whose bg is kept low by low carbs alone. And although the latter situation is a good thing while it lasts, I have seen reports that long term that fat may damage beta cells permanently and take away the diet-only option of good T2 management. So I believe that carb reduction has a powerful role to play but that the fat stripping is a greater priority and is not getting enough emphasis.
This is interesting. I managed by low carbing for many years but now that is no longer working and I do think my beta cells have declined. I take glimepiride now. In the past I have had a cut off point with my weight, below which my Hba1c was very good. Unfortunately I went above the threshold and couldn't lose the weight no matter what I tried.I believe it takes only half a gram of fat in the pancreas to compromise the insulin signalling or secretion, so if one’s (dratted!) genes are disposed to direct excess fat to the pancreas rather than subcutaneously then that’s how easy it is for a little weight gain to undo our remission. My concern is that there may be people who have not expelled that fat but whose bg is kept low by low carbs alone. And although the latter situation is a good thing while it lasts, I have seen reports that long term that fat may damage beta cells permanently and take away the diet-only option of good T2 management. So I believe that carb reduction has a powerful role to play but that the fat stripping is a greater priority and is not getting enough emphasis.
Can you provide a reference or link to these figures please?My understanding is that if one’s initial weight loss has been sufficient to clear excess fat from the pancreas and so get one’s A1c down into the remission zone then allowing weight to rise by as little as 6 lbs can be sufficient to put one back into non-remission.
The figure of about 3kg regain as a sufficient trigger for loss of remission is one I picked up from several papers a couple of years back and kept in my memory, but I do not have all the refs readily to hand now. But it’s a threshold figure that Taylor has often mooted from his own experience as stated near the end of the article below where he says that sometimes “a few kilograms” can be enough.Can you provide a reference or link to these figures please?
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