To make more of the results as presented by
@LittleGreyCat we probably need to compare the figures and graphing to a non-diabetic as i suspect that the insulin rise is later than would be normal ( normal as in a non-diabetic) and that this mistiming is part of the problem which would likely become more apparent if the glucose/ blood sugar rise amount from the meal was high enough.
Conversely the lower the stimulus from carb intake to better.
Noting that the test was a fasting one. the results should indicate how the body is coping in the absence of food and without the post meal stimulus for insulin production.
I read it as my body maintaining a higher than optimum BG level in a fasted state.
Possibly due to a miscommunication between the pancreas and the liver.
It could be the pancreas isn't producing enough insulin to match the BG level.
It could be that the liver isn't responding to the insulin and is continuing to release glucose into the bloodstream.
Edit: Aaaargh! Misread what glucagon does. Following bit revised.
It could be that the Alpha cells in the pancreas are releasing too much glucagon to boost the release of glucose by the liver or the liver is not responding to the glucagon correctly.
Hmmmm.....that is an interesting one!
Beta cells release insulin to get glucose out of the blood stream.
Alpha cells release glucagon to stop the BG level falling and get liver releasing glucose into the bloodstream.
I wonder if Alpha cell dysfunction could lead to dawn phenomenon and slow insulin response?
I also wonder if the pancreas switches from insulin to glucagon production in a synchronised manner and "assumes" that the liver will do as it is told? Which could result in lower insulin levels and higher BG levels in a fasting state.
Or that glucagon is released and then the BG overshoots because the liver is not listening to the insulin?
Or.......
Whatever, the main discussions seem to be about Beta cells, which are only one half of the balancing mechanism.