NoCrbs4Me
Well-Known Member
- Messages
- 3,700
- Location
- The Rocky Mountain Foothills, Canada
- Type of diabetes
- I reversed my Type 2
- Treatment type
- Other
- Dislikes
- Vegetables
He's a pretty smart guy. I wouldn't dismiss his ideas too casually.Have you read his recent articles on what causes heart disease? I think he is rather enamoured of his own wisdom and has ignored the science behind diabetes and heart disease? D.
He's a pretty smart guy. I wouldn't dismiss his ideas too casually.
I seldom agree with GPs. I didn't say you should believe Kendrick because he is smart or a GP. I didn't even say you should agree with him or even that I agreed with him. Rather, I suggested that because he is smart, his ideas shouldn't be casually dismissed. You can believe or not believe whatever you want to, I suppose, but I think Kendrick makes some persuasive arguments. If you haven't read his book, you should before passing final judgement.My eldest is a long standing GP and I don't always agree with her! Not even the Pope is always right!
Hi, I see Dr. Kirpich has also tried diets for diabetics and obesity involving 40%carb and the rest protein and unsaturated (in the main) fats. It is not surprising they lost weight since the bda and the American dietetic association recommend a barmy 60% carbs! Her patients would have lost more if she had tried a really low carb high good fats diet which many of us have test proved already. BTW Does anyone know which probitics improve the gut biome or whatever it's called? thanks regards D.
Well, I don't know if probiotics will help with your liver, but I got rid of my self-diagnosed NAFLD without probiotics. I just ate very little carbs and lots of animal fat.
Spendercat, When you have had an Hb1Ac test done, ALWAYS ring the surgery and ASK FOR A PRINTED COPY OF THE RESULTS! This shows you what tests were done, what the target figures for each test is, and what your reading was. Regarding "Fatty Liver", the target on test XaLJx is 7 to 40. mine is currently 42, so just outside range. On your results sheet Fatty Liver is called "Serum Alanine Aminotransferase Level". Remind yourself not to accept a standard "Satisfactory" over the phone but collect a print out and examine it (or turn it into a Results Spreadsheet to monitor your progress, and give you questions to discuss with your GPHow do people know they have this? Doesn't everyone who is overweight have it?
Does it actually do any harm? More than diabetes that is.
Spendercat, When you have had an Hb1Ac test done, ALWAYS ring the surgery and ASK FOR A PRINTED COPY OF THE RESULTS! This shows you what tests were done, what the target figures for each test is, and what your reading was. Regarding "Fatty Liver", the target on test XaLJx is 7 to 40. mine is currently 42, so just outside range. On your results sheet Fatty Liver is called "Serum Alanine Aminotransferase Level". Remind yourself not to accept a standard "Satisfactory" over the phone but collect a print out and examine it (or turn it into a Results Spreadsheet to monitor your progress, and give you questions to discuss with your GP
some things in this theory don't make sense to me. Like why are the bad guys so strong and vigorous and the good ones wilt and die easily. Why are the good ones so feeble that they need probiotics every day but the bad guys don't need that to thrive.I'm extremely skeptical of the gut biome theory.
some things in this theory don't make sense to me. Like why are the bad guys so strong and vigorous and the good ones wilt and die easily. Why are the good ones so feeble that they need probiotics every day but the bad guys don't need that to thrive.
...Because the anti-inflammatory effects were observed in both the presence and absence of the antisteatotic effects, our findings further suggest that the anti-inflammatory property of rFGF1 is independent of its effect on lipid catabolism. Our current findings indicate that, in addition to its potent glucose-lowering and insulin-sensitizing effects, rFGF1 could be therapeutically effective in the treatment of NAFLD.
http://www.nature.com/onc/journal/v22/n28/full/1206499a.htmlSeveral growth factors have been suggested to play a crucial role in liver regeneration, but a functional proof is still missing. Since fibroblast growth factors are important for the initiation of mammalian liver development, we determined the roles of these mitogens in liver repair by targeted expression of a dominant-negative fibroblast growth factor receptor (FGFR) in hepatocytes of transgenic mice. The liver of young animals appeared histologically normal, and liver function was not obviously impaired. In aged transgenic mice, the frequency of fatty liver development was strongly increased compared to control animals. Following partial hepatectomy, transgenic mice showed markedly reduced hepatocyte proliferation because of an arrest in the late G1 phase of the cell cycle. These data demonstrate a key role of FGFR signalling in repair after liver injury.
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