Strictly speaking Fasting Blood Glucose must be tested at least 8 hours after eating, and not drinking anything other than water during that time. Testing first thing in the morning before breakfast is the most convenient time, though it doesn't really matter how long you've been asleep so long as you haven't eaten for at least 8 hours.
Fasting Blood Sugar Levels
Fasting, as the name suggests, means refraining from eating of drinking any liquids other than water for eight hours. It is used as a test for diabetes.www.diabetes.co.uk
The idea is to measure blood glucose at a time when food intake does not distort the numbers. In the fasted state blood glucose levels are almost entirely controlled by the pancreas and liver.
In simplistic terms, when fasting:
1: The pancreas produces glucagon when blood glucose levels get low
2: The liver responds to glucagon by producing glucose
3: The pancreas responds to sufficient glucose levels by stopping glucagon production and by producing insulin
4: The liver responds to insulin by stopping glucose production
5: This cycle repeats, over and over, to keep blood glucose levels quite stable
In a Type 2 diabetic steps 3 and 4 tend to be compromised. Again, in simplistic terms, the pancreas doesn't produce enough insulin and/or the liver doesn't respond to insulin properly. The result is higher fasting blood glucose levels.
All what you say is quite factual and happens as you say.Strictly speaking Fasting Blood Glucose must be tested at least 8 hours after eating, and not drinking anything other than water during that time. Testing first thing in the morning before breakfast is the most convenient time, though it doesn't really matter how long you've been asleep so long as you haven't eaten for at least 8 hours.
Fasting Blood Sugar Levels
Fasting, as the name suggests, means refraining from eating of drinking any liquids other than water for eight hours. It is used as a test for diabetes.www.diabetes.co.uk
The idea is to measure blood glucose at a time when food intake does not distort the numbers. In the fasted state blood glucose levels are almost entirely controlled by the pancreas and liver.
In simplistic terms, when fasting:
1: The pancreas produces glucagon when blood glucose levels get low
2: The liver responds to glucagon by producing glucose
3: The pancreas responds to sufficient glucose levels by stopping glucagon production and by producing insulin
4: The liver responds to insulin by stopping glucose production
5: This cycle repeats, over and over, to keep blood glucose levels quite stable
In a Type 2 diabetic steps 3 and 4 tend to be compromised. Again, in simplistic terms, the pancreas doesn't produce enough insulin and/or the liver doesn't respond to insulin properly. The result is higher fasting blood glucose levels.
Agreed - I left out all the complexity as I don't understand it and can't ever hope to. I wasn't aware that the first phase insulin response occurred in the absence of a rapid rise in blood glucose as a result of a meal for example. I had assumed that there was a gentle 'basal' insulin response in the fasted state, which goes to show how little I know on the subject.All what you say is quite factual and happens as you say.
But have missed important details such as the first and second insulin (hormonal) response....
My condition goes against many specialist theories about the science of how and why, the hypos happen because my BG levels go above a certain level, somewhere above 7mmols ish. That is the trigger for the signals to overproduce insulin because the first phase wasn't enough. It has a lot to do with cell production of certain cells, indeed epinephrine is one. The science thing is complicated. But the process of you leave out the details is comprehensive explained. And that is what I had to discover, it was quite rare when I was diagnosed, but as the condition is becoming more understood by more specialist endocrinologists.Agreed - I left out all the complexity as I don't understand it and can't ever hope to. I wasn't aware that the first phase insulin response occurred in the absence of a rapid rise in blood glucose as a result of a meal for example. I had assumed that there was a gentle 'basal' insulin response in the fasted state, which goes to show how little I know on the subject.
I doubt any of us on this forum will understand all the complexities in our lifetimes. Firstly it's fiendishly complicated, and secondly the science isn't there yet even if we wanted to learn it all. The 'fasting hormone' asprosin, which might perhaps (in my amateur opinion) be part of the explanation for the dawn phenomenon, was only discovered in 2016 for example. Who knows what other pieces of the puzzle remain to be discovered, and how long it will be before some genius manages to fit all those pieces together and explain it all.