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How an underactive thyroid affects diabetes
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<blockquote data-quote="DCUKMod" data-source="post: 2228879" data-attributes="member: 345386"><p>That's all very well, until one develops thyroid issues when A1c is down to around 30, low BMI, HOMA-IR of around 1, and having been eating LC for a number of years.</p><p></p><p>Many Endos do not favour low carb, let along keto for those with hypothyroidism, as the claim is reduces T3 performance. My Endo swims against the tide on that bit.</p><p></p><p>Sadly, thyroid dysfunction is just like metabolic dysfunction; it has little hard and fast about it, and only digging deep, on a personal level will begin to give the clues to the puzzle.</p><p></p><p>As for symptoms from the list above, I was:</p><p></p><p>fatigue; a little</p><p>increased sensitivity to cold; a lot - this was my main issue</p><p>constipation; plenty, but longer standing than any other thyroid symptoms</p><p>dry skin; i could be persuaded, but then skin tends to dry as we age </p><p>weight gain; none - 1-1.5kg at the absolute maximum </p><p>puffy face; No</p><p>hoarseness; No</p><p>muscle weakness; No</p><p>elevated blood cholesterol level; yes, but longer standing that any hypothyroid signs</p><p>muscle aches, No</p><p>tenderness and stiffness; No </p><p>pain, Only related to gluten intolerance </p><p>stiffness or swelling in your joints; Marginal - long standing osteoarthritis in my hands</p><p>heavier than normal or irregular menstrual periods; no idea - post-hysterectomy </p><p>thinning hair; yes, but only latterly as my condition deteriorated</p><p>slowed heart rate; No</p><p>depression, low mood, but related to the frustration of the temperature regulation issues</p><p>impaired memory, In periods</p><p>enlarged thyroid gland (goiter). - No</p><p></p><p>As you can see, I am atypical in a million ways, but then I was always atypical T2.</p><p></p><p>To be fair, I haven't watched the video, but if he relies on TSH as any form of marker, after 1 or 2 tests, he is doing his patients a massive disservice. TSH signals the pituitary to release thyroxine (T4), it gives no indication whatsoever whether the pituitary actually produces the passive T4, or whether the individual can convert any T4 they produce into the active T3.</p><p></p><p>It's a bit like relying on random blood glucose tests to manage diabetes.</p></blockquote><p></p>
[QUOTE="DCUKMod, post: 2228879, member: 345386"] That's all very well, until one develops thyroid issues when A1c is down to around 30, low BMI, HOMA-IR of around 1, and having been eating LC for a number of years. Many Endos do not favour low carb, let along keto for those with hypothyroidism, as the claim is reduces T3 performance. My Endo swims against the tide on that bit. Sadly, thyroid dysfunction is just like metabolic dysfunction; it has little hard and fast about it, and only digging deep, on a personal level will begin to give the clues to the puzzle. As for symptoms from the list above, I was: fatigue; a little increased sensitivity to cold; a lot - this was my main issue constipation; plenty, but longer standing than any other thyroid symptoms dry skin; i could be persuaded, but then skin tends to dry as we age weight gain; none - 1-1.5kg at the absolute maximum puffy face; No hoarseness; No muscle weakness; No elevated blood cholesterol level; yes, but longer standing that any hypothyroid signs muscle aches, No tenderness and stiffness; No pain, Only related to gluten intolerance stiffness or swelling in your joints; Marginal - long standing osteoarthritis in my hands heavier than normal or irregular menstrual periods; no idea - post-hysterectomy thinning hair; yes, but only latterly as my condition deteriorated slowed heart rate; No depression, low mood, but related to the frustration of the temperature regulation issues impaired memory, In periods enlarged thyroid gland (goiter). - No As you can see, I am atypical in a million ways, but then I was always atypical T2. To be fair, I haven't watched the video, but if he relies on TSH as any form of marker, after 1 or 2 tests, he is doing his patients a massive disservice. TSH signals the pituitary to release thyroxine (T4), it gives no indication whatsoever whether the pituitary actually produces the passive T4, or whether the individual can convert any T4 they produce into the active T3. It's a bit like relying on random blood glucose tests to manage diabetes. [/QUOTE]
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