bulkbiker
BANNED
- Messages
- 19,575
- Type of diabetes
- Type 2
- Treatment type
- Diet only
Think it was the raw fruitarianism that did for him in the end.. all that fructose going to the liver..Ha ha, yes, good! A bit like how the Steve Jobs' diet story that showed up yesterday in the news when my friend told me she had tried Tim Ferris' Slow Carb diet but then found herself craving sweet so she would eat 4 apples with her coffees.
https://www.afr.com/life-and-luxury...hat-included-fasting-for-days-20190729-p52bn1
@Cocosilk I’m interested in why you think carb restriction increases insulin resistance. Could you say a bit more?
I don't understand the question.
If you are eating keto then you aren't eating carbs in quantities to cause damaging spikes.
Therefore your question is redundant.
Keto is, quite simply, avoiding carbs to the point that the body runs on ketones.
Therefore no 'damaging carb spikes' will happen.
I think the first I heard about the temporary insulin resistance you will experience while eating very low carb was when I was preparing to do a follow up glucose tolerance test. They tell you to make sure you have eaten at least 150g of carbs per day for the 3 days leading up to the test otherwise your results won't be reliable.
I think I messed my GTT up because I only ate a few extra carbs (pancakes) for 2 days prior instead of 3.
This article also talks about the kind of insulin resistance you get while low carb eating. https://www.dietdoctor.com/low-carb/fasting-blood-glucose-higher
I assume though that this should go back to whatever your normal is after 3 days of reintroducing carbs again based on how to prepare for a GTT.
For me the problem is I'm not sure if I'm still a bit more insulin resistant than I should be because I actually have a glucose intolerance and not just because of keto/low carb eating.
Thanks for that. I think the key phrase from that article is:
‘The scientific name is “physiologic insulin resistance” and it’s a good thing — unlike “pathologic insulin resistance.” ‘ (There is a longer explanation of what that means, but in essence a keto diet is improving the type,of insulin resistance that underpins Type 2, not worsening it.
I don’t understand the assumptions made by Jason Fung regarding physiological insulin resistance and adaptive glucose sparing. Jason Fung frequently says that the the body isn’t stupid. Why would the fasting blood sugar run higher if the body is running on fat like in keto? (One would think the body would keep tight control on the glucose and not have more in the blood than needed. Less should be needed when one is burning fat.)
@Cocosilk
I think you may be confusing the last meal effect with physiological insulin resistance
Last Meal Effect aka known as the https://en.m.wikipedia.org/wiki/Staub-Traugott_Phenomenon is where the body downgrades insulin production (or increases it) According to the amount of insulin required to deal with recent meals. This is what causes spikes after an unexpectedly carby meal. It can take a few days for the body to wake up to the fact that more carbs are coming in regularly now, and until that happens, the person experiences bg spikes after carbs. The intake of carbs/spikes means that they are extremely unlikely to be in ketosis.
Physiological Insulin Resistance is the glucose sparing bit, where people who have been in nutritional ketosis for months, tend to find that their fasting blood glucose rises a bit, and their overall blood glucose levels stay very stable. It only happens in the consistent absence of carbs, and if carbs are reintroduced to the diet, the Last Meal Effect will kick in, insulin production will increase, and the individual will lose their PIR, probably resulting in somewhat lower fasting bgs and more fluctuation in bgs through the day, due to the carbs.
Yes, I agree with @Brunneria
I had a bout of physiological insulin resistance a few years ago now. What happened to me was my base line levels increased, fasting, before meals and bedtime, but the rises post meal stayed the same as before. If I had a meal that would see me rise by 1.5mmol/l before the PIR , the same meal would also see a similar rise, but the levels would be higher because I started higher.
It lasted a few weeks, then disappeared, never to return.
I don't understand the question.
If you are eating keto then you aren't eating carbs in quantities to cause damaging spikes.
Therefore your question is redundant.
Keto is, quite simply, avoiding carbs to the point that the body runs on ketones.
Therefore no 'damaging carb spikes' will happen.
Can we please get back to discussing the title of this thread now?
Do we have to? I love the different tangents. I often find I learn something new when someone comes in with a comment like LittleGreyCat's.Can we please get back to discussing the title of this thread now?