But surely, if this is correct and, as stated it continues throughout the disease process, then how can the medical profession sign someone off the register when in non-diabetic numbers? Surely something isn't right there?β-Cell function decline: the major cause of disease progression
A hallmark of type 2 diabetes is a decline in β-cell function, which begins as early as 12 years before diagnosis and continues throughout the disease process. https://care.diabetesjournals.org/content/32/suppl_2/S151
I tend to agree with you. Bilous does mention things such as drug-induced diabetes (in the table I didn't type out) but these exemptions don't appear to require a change to the sweeping nature of his definition.And that definition would include T1 and T2 but would not include me as I have drug induced diabetes I don't think it's possible to define diabetes as a single entity..
As I pointed out in the other thread you were posting in, the method used by Prof Taylor to show insulin responses includes BSA and hence body weight. So the insulin response will appear to improve simply because it was a weight loss diet and is a mathematical consequence of the weight term diminishing. Those figures are misleading and the claim cannot be held up as viable evidence.Recovery of first-phase insulin response (0.04[−0.05–0.32] to 0.11[0.0005–0.51] nmol/min/m2, p < 0.0001) defined those who returned to non-diabetic glucose control and this was durable at 12 months (0.11[0.005–0.81]
https://www.sciencedirect.com/science/article/pii/S1550413118304467
What proportion? Do you have a link?@Tannith yet again I’d ask how does the theory (it’s not proven) you advocate stand up in light of insulin t3sts that show a large proportion of type 2 overproduce insulin if their beta cells are defunct?
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6735759/What proportion? Do you have a link?
thank you for this link.
Oddly as a low carber I’m not totally adverse to the idea of a personal fat threshold - for some T2 it seems valid. However….This seems like a good place to post this - a long interview with Prof. Roy Taylor covering his 'Twin Cycle Hypothesis' as the root cause of Type 2, his (I think it's his?) 'Personal Fat Threshold' theory, the role of genetics, and lots of other interesting stuff.
I'm not sure I agree with everything he says here, and the low-carbers won't agree with his attitude on that front, but he certainly makes some compelling arguments.
I thought T2s had high insulin amounts (unless very long term)?My C-peptides are low/normal.
Have you been checked for this ? Low cpeptide and sibling with LADA sound like red flags to me. (Disclaimer, am not a doctor).excluding my brother who has LADA
I know and I hear you. I'm already living with one autoimmune condition. It seems that while I'm controlling my blood sugars through diet and exercise my T2 diagnosis will remain. My Dr is all over the very low carb diet and exercise gig and is encouraging me to stick with it. So I guess while I am controlling my blood sugar it will be a wait and see game.I thought T2s had high insulin amounts (unless very long term)?
Have you been checked for this ? Low cpeptide and sibling with LADA sound like red flags to me. (Disclaimer, am not a doctor).
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