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It is just my preference to be below 40.Below 6 is normal, why would you be disappointed with a normal response?
http://www.diabetes.co.uk/what-is-hba1c.html
It is just my preference to be below 40.Below 6 is normal, why would you be disappointed with a normal response?
http://www.diabetes.co.uk/what-is-hba1c.html
Hi@yetta2mymom - diabetes has not been pinned down to one single gene. The latest science on epigenetics seems to indicate a much higher degree of complexity than simply one gene = one condition. Genes can be switched on or off by environmental factors, diet, etc. I recently had my DNA sequenced and the report came back with 20 different SNP's indicating risk of Type 2, but I still don't think it was inevitable that I would go on to get it. I also have really high risk factors for HLA-B27 syndromes (like psoriasis) but (touch wood) haven't developed signs of any of them yet. My mother on the other hand was a life-long severe psoriasis sufferer. Who knows why she got it but I haven't so far?
HiIt is just my preference to be below 40.
As you ask, I would personally be disappointed with a 5.8 A1C despite a severe Atkins diet if I were non-diabetic.
It is just my preference to be below 40.
It's not a competition. I am happy with my results and targets, thanks.40 is actually 5.8%, and the tolerance, and personal hemoglobin means you could actually be over, so you may need to re-assess your own targets.
Who knows, the op may actually have better control than yourself.
It's not a competition. I am happy with my results and targets, thanks.
The opening post asked if 5.8 (40) was an expected result after 7 years of strict Atkins. I said would personally be disappointed.I would guess the op is, why did you bring it 40 up as a problem, if it's your personal target as well?
Can I remind you, the ethos on here is to be nice, especially to newbies?
40 is actually 5.8%, and the tolerance, and personal hemoglobin means you could actually be over, so you may need to re-assess your own targets.
Who knows, the op may actually have better control than yourself.
I really hope you are producing insulin. If you aren't making insulin and you aren't injecting it, you will die.
There will be a steady stream of glucose going into your blood from your liver - your body will be making insulin to get that energy into your cells. Even those who are keto adapted need insulin (sure a type 1 in nutritional ketosis will be having significantly less insulin, but they will still be having some insulin, just like someone who doesn't rely on exogenous insulin will still be producing insulin on a severe Atkins diet).
Maybe it would help your understanding and development of your theories if you had a cpeptide test to see what your insulin production is like? Just a thought.
Good thought, but I have no time.
Also, propagating the idea that you can live without insulin is, frankly, dangerous - either you are making it or you are injecting it or you are dead. Those are the only options. A type 1 on a low carb diet with no injections will be getting away without injections because they are honeymooning and thus producing some of their own insulin.
I really hope you are producing insulin. If you aren't making insulin and you aren't injecting it, you will die.
There will be a steady stream of glucose going into your blood from your liver - your body will be making insulin to get that energy into your cells. Even those who are keto adapted need insulin (sure a type 1 in nutritional ketosis will be having significantly less insulin, but they will still be having some insulin, just like someone who doesn't rely on exogenous insulin will still be producing insulin on a severe Atkins diet).
Maybe it would help your understanding and development of your theories if you had a cpeptide test to see what your insulin production is like? Just a thought.
Also, propagating the idea that you can live without insulin is, frankly, dangerous - either you are making it or you are injecting it or you are dead. Those are the only options. A type 1 on a low carb diet with no injections will be getting away without injections because they are honeymooning and thus producing some of their own insulin.
Hi
I am probably pretty close to a type 1 diabetic on my diet (maybe a little more insulin). Just for your information I have been on a severe Atkins style diet for about 7 years.
Hi
I am probably pretty close to a type 1 diabetic on my diet (maybe a little more insulin). Just for your information I have been on a severe Atkins style diet for about 7 years. With my "hunter" gene I have at first lost then gained back weight (I am fat). I am now trying to cut the calories and lose some weight. I note that with low sugar my experience, if I try and lose weight, is my body can go into save energy mode. It may have. Strangely I may make me half awake but if I eat enough sugar losing weight does not lead to this effect. I am resting/sleeping too many hours each day. Yes my sleep is disturbed but not more than most people my age (77) and one effect of my autoimmune episode is I wake during most rem dreams. No, the memory of the dream is usually quickly gone and usually I am back asleep within a few minutes.
This is really the heart of the matter. Unless you are T1 and producing NO insulin there is insulin present at some level. The pancreas does not come with a tap you can turn on and off. Very low carb reduces the amount of insulin needed to handle the carb load but does not eliminate the need entirely. As for the body and brain using up what little glucose is in the blood, it needs insulin to do that. And I totally agree that giving someone who is a brand new type 1 the idea that they could just change their diet and stop using insulin is dangerous.
@yetta2mymom I wish you luck in finding the information and doctor you are looking for, but I think it is becoming apparent to me that you aren't going to find it on this thread. It sounds like you are looking for a single doctor who has made a single statement at some point in the past 6 decades, about this so called hunter gene. It is my (uneducated) guess, that this is a phrase only he used, and you are going to struggle to find it.
If I was as desperate as you clearly are to find an answer, I would be grabbing at each and every insight that has been thrown at you on this thread, and be spreading my energy across them all.
"The best way to show that a stick is crooked is not to argue about it or to spend time denouncing it, but to lay a straight stick alongside it." D.L.Moody
I hope you find what you are looking for.
@yetta2mymom I wish you luck in finding the information and doctor you are looking for, but I think it is becoming apparent to me that you aren't going to find it on this thread. It sounds like you are looking for a single doctor who has made a single statement at some point in the past 6 decades, about this so called hunter gene. It is my (uneducated) guess, that this is a phrase only he used, and you are going to struggle to find it.
If I was as desperate as you clearly are to find an answer, I would be grabbing at each and every insight that has been thrown at you on this thread, and be spreading my energy across them all.
"The best way to show that a stick is crooked is not to argue about it or to spend time denouncing it, but to lay a straight stick alongside it." D.L.Moody
I hope you find what you are looking for.
@yetta2mymom I have read your posts and I really think that this 'hunter gene' you're referring to is, as suggested before, simply another name for the 'thrifty gene'.
Have a look at this article:
https://sciencelife.uchospitals.edu/2013/03/07/how-type-2-diabetes-survived-evolution/
" A popular explanation is that insulin resistance, a predisposition to type 2 diabetes, actually provided a protective benefit to humans who lived as hunter-gatherers. Insulin resistance limits the body’s intake of glucose by muscle and liver cells, therefore allowing its storage by fat cells. The body could use this extra glucose, so the theory goes, during cycles of feast and famine when people couldn’t be sure of their next meal. This so-called “thrifty genotype” hypothesis was proposed by University of Michigan geneticist James Neel in 1962, and still holds a lot of sway in the scientific community.
Another similar explanation, called the “carnivore connection,” is that insulin resistance developed in hunter-gatherers and herders because they had a low-carbohydrate (and low glucose), protein-rich diet, resulting in the need to save glucose in the blood to make it accessible for the brain. This same resistance was no longer needed in early farmers, however, because they had begun to incorporate carbohydrates into their diets as they learned to cultivate grains"
So the 'hunter' has something very similar to the thrifty gene. Other sites have posited similar things - that some people are better suited to a lower carb diet.